近年来，在PD CD 4基因对程序性细胞死亡的影响，以及对转化和蛋白质翻译的抑制作用的相互关系等方面进行了大量研究。
Quantity of studies of PDCD4 have been carried out about its effect on programmed cell death and correlationship with inhibition for transformation and protein translation in recently year.
活性氧可以诱导细胞程序性死亡或坏死、可以诱导或抑制许多基因的表达，也可以激活上述级联信号。
Ros induce programmed cell death or necrosis, induce or suppress the expression of many genes, and activate cell signalling cascades, such as those involving.
结论：稳定表达人ox40l的乳腺癌转基因细胞在体外能有效地活化t细胞，介导其增殖、分泌细胞因子及抑制T细胞活化诱导的细胞死亡。
CONCLUSION: OX40L-MDA-MB-435 cells could activate t cells in vitro, promote t cell proliferation and cytokine secretion, and suppress t cell activation-induced cell death.
结论：稳定表达人ox40l的乳腺癌转基因细胞在体外能有效地活化t细胞，介导其增殖、分泌细胞因子及抑制T细胞活化诱导的细胞死亡。
CONCLUSION: OX40L-MDA-MB-435 cells could activate t cells in vitro, promote t cell proliferation and cytokine secretion, and suppress t cell activation-induced cell death.
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