单灶性 CI患者的梗死体积与血清胆固醇水平相关（P <0.01）。
The infarction volumes of unifocal CI patients was associated with the serum cholesterol level (P<0.01).
黄体酮；脑梗死；白介素6；梗死体积。
主要观察指标：模型出现率；梗死体积。
MAIN OUTCOME MEASURES: Model successful rate and infarction volume.
观察脑梗死体积、神经功能缺损评分和病理改变。
局灶脑缺血40分钟后再灌注时进行局部亚低温能减轻脑梗死体积。
Local mild hypothermia could decrease the volume of cerebral infarct, reperfusion following local ischemia for 40 minutes.
晚期6小时纤溶酶原激活剂没有减少梗死体积，而是更坏出血性转换。
Late 6-hour tPA did not decrease infarction but instead worsened hemorrhagic conversion.
结论缺血性脑卒中患者急性期血小板活化增强，且与脑梗死体积相关。
Conclusion the activation function of platelet in the acute phase of patients with ischemic stroke are increased, and it is associated with the volume of cerebral infarction.
目的：研究脑梗死大鼠在不同电刺激治疗后血管构筑及梗死体积的变化。
AIM: To investigate blood vessel build and infarcted area of rats after stroke by different electric stimulation.
西比灵组脑梗死体积比明显小于缺血再灌注组（P<0 .0 5 ）。
The ratio of infarct volume of group using sibelium was smaller than that of ischemic reperfusion group( P< 0 05).
血清MMP-2和CRP与脑梗死体积、患者神经功能缺损程度呈正相关。
There were obvious positive correlations of MMP-2 and CRP levels to volume of infarct and degree of neurofunction impairment.
对照组两次均为假手术，比较各组神经功能评分、梗死体积及NGF的表达。
Neurologic score , infarct volume and the expression of NGF were compared in each group.
运用神经功能缺损评分和焦油紫染色观察动物的神经功能症状与脑梗死体积；
Neurological deficit score and infarct volume in all groups were compared to estimate ischemic injury.
戒烟组神经功能评分及梗死体积明显低于吸烟4周组，与对照组无显著差别。
Stop smoking group were significantly reduced compared with the smoking-4 week group and there were no significant difference between the stop smoking group and control group.
结论：脑梗死急性期血清mmp - 9水平可能与梗死体积和近期预后密切相关。
Conclusion: Serum MMP-9 levels may be closely associated with the infarction volumes and the recent prognosis in acute cerebral infarction.
目的研究先兆tia对其后脑梗死体积和临床严重度的影响，探讨TIA的临床意义。
Objective To explore the effect of prodromal transient ischemia attack (TIA) on infarction volume, clinical severity and prognosis in patients with subsequent cerebral infarction.
在以后的研究中，我们将延长实验观察时间，以便更准确的评价移植细胞对梗死体积和神经功能的作用。
In the coming research, we'll prolong the observation time, hoping to evaluate the influence of Transplanted cells on the bulk of infarct and the neurological function.
结果：三七总皂苷可有效降低脑缺血再灌注损伤大鼠的脑梗死体积，明显减轻脑缺血区血脑屏障破坏的程度。
Result: PNS could reduce infarct volume of cerebral ischemia-reperfusion injury in rats, reduce the extent of the damage in blood-brain barrier significantly.
方法采用线栓法制作大鼠局灶性脑缺血再灌流模型，大脑中动脉阻塞2小时，再灌流4小时后观察梗死体积的变化。
Methods We made infarct models of rats with thread embolism and observed the changes of the infarct volume in rats which had re perfused 4 hours after 2 hours embolism.
MBP的血浆浓度与梗死体积呈显著正相关（P<0.01），与神经功能缺损程度也呈显著正相关（P<0.01）。
The release of MBP in patients was associated with the volume of brain lesions (P<0.01) and the neurological outcome as assessed by NIHSS (P<0.01).
测体积组将鼠脑经TTC染色后，用计算机病理图象分析仪测量出梗死面积，根据梯形法则计算出梗死体积及梗死体积比。
The rat brains of the group measured volume were dyed with TTC and measured the area by computerized pathological image analyzer.
通过免疫组织化学方法和原位末端标记法（TUNEL）检测热休克蛋白70表达及凋亡细胞数，TTC染色观察梗死体积。
The expression of heat shock protein 70 was examined by immunohistochemistry, the apoptosis of neurons by TUNEL and the volume of cerebral ischemia by TTC staining.
HE染色及免疫组化检测脑梗死灶体积、病理改变及CD34阳性细胞浸润情况。
The technique of TTC and HE staining and immunohistochemistry were used to detect the volume of infarction and pathological change and the infiltration of CD34 positive cells, respectively.
尽管在本研究中未发现梗死灶体积差异，但本研究结果提示SDF - 1与其受体的作用促进移植细胞向损伤区的迁移。
Although we didn't find any differences among infarcts, we do find that SDF-1 and its receptor can contribute to Transplanted cells' migration to the damage area.
尽管在本研究中未发现梗死灶体积差异，但本研究结果提示SDF - 1与其受体的作用促进移植细胞向损伤区的迁移。
Although we didn't find any differences among infarcts, we do find that SDF-1 and its receptor can contribute to Transplanted cells' migration to the damage area.
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