血管密度高的核团包括了脑桥核、下橄榄核簇等等。
The high density nuclei consist of the pontine nuclei, inferior olivary complex and so on.
他们发现呼吸中枢的刺激来自于侧脑桥网状结构和侧位的下行神经通路。
They found the stimulation of the respiratory center by the lateral pontile reticular formulation and laterally located descending neural pathway.
大脑做梦的关键区域不是脑桥，而是新皮层中位于及靠近枕叶交叉部分的视觉区域和视听区域。
The regions critical for dreaming are not in the pons. They include the visual and audiovisual regions in and near the temporoparietal-occipital junction in the neocortex.
当病人大脑脑干的“脑桥”部分被废，快速眼部活动(REM)睡眠不再出现。
When a part of the brain stem known as the pons is destroyed, people no longer experience REM sleep.
后脑位于大脑后端的下方，由小脑、脑桥和延髓构成。
The hindbrain sits underneath the back end of the cerebrum, and it consists of the cerebellum, pons, and medulla.
结论脑桥腔隙性梗死占脑桥梗死的绝大多数且预后较好。
Conclusion pontine lacuna infarctions occupy most of the pontine infarctions and its prognosis is well.
组织，细胞质蛋白，病人，老年性痴呆，脑，桥脑。
Tissue, Cytoplasmic Protein, Human Disease, Alzheimer's Disease, Brain, Pons.
组织，膜蛋白，病人，老年性痴呆，脑，桥脑。
Tissue, Membrane Protein, Human Disease, Alzheimer's Disease, Brain, Pons.
结论脑桥梗死多发生在脑桥中上部的旁正中区域，主要由椎基底动脉狭窄、高血压性基底动脉穿通支闭塞所致。
Conclusions pontine infarctions, which mainly located in mid-upper paramedian pons, are usually due to vertebrobasilar artery stenosis and occlusion of pontine perforating arteries by hypertension.
组织，总蛋白， 病人，老年性痴呆，脑，桥脑。
Tissue, Total Protein, Human Disease, Alzheimer's Disease, Brain, Pons.
组织，核蛋白，病人，老年性痴呆，脑，桥脑。
Tissue, Nuclear Protein, Human Disease, Alzheimer's Disease, Brain, Pons.
目的分析酷似大脑半球病变的脑桥梗死的临床和影像学特点、定位错误的原因。
To investigate clinical presentation of pontine infarctions mimicked cerebral hemisphere infarctions, and to analyse the causes of bias in localization diagnosis.
结论脑桥梗死多数为腔隙性梗死，其临床表现与病灶部位及大小有关。
Conclusion Pontine infarctions were mostly lacuna infarctions and their clinical manifestation was related to the position and size of infarction focuses.
在30处中脑-间脑接合处病灶中，11处病灶显著向上延伸累及间脑结构，18处病灶显著向下延伸累及脑桥延髓区域。
Of the 30 MDJ lesions, 11 showed a marked upward extension involving the diencephalic structures and 18 had a prominent downward extension involving the pontobulbar region.
病报部位多在肤阱脸体、脑桥、小脑茎中部、颅骨圈突部。
Sickness newspaper spot many middle the skin pitfall face body, pons, cerebellum stem, skull circle suddenly.
次常见的病灶部位分布于脑桥延髓区，约40%的患者可见此改变。
The next most common location of involvement was the pontobulbar region, seen in 40% of the cases.
脑桥、小脑和延髓组成了后脑。
The pons the cerebellum and the medulla oblong ata composed the hindbrain.
这些神经细胞位于脑干的脑桥部位。
目的对经中颅窝入路小脑脑桥角区的显露程度及其损伤程度进行定量综合评价。
Objective To evaluate comprehensively the exposure and damage to the cerebellopontine angle region by the middle cranial fossa approach.
本实验用HRP逆行追踪和免疫细胞化学结合法研究了大鼠脑桥内GABA能神经元至小脑前叶皮质的投射。
Projections from GABA-ergic neurons of the pons to the cortex of anterior cerebellar lobe have been studied by combined method of HRP retrograde tracing and immunocytochemistry.
随着CT的临床应用，原发性脑桥出血的发现率明显提高。
More and more cases of primary pontine hemorrhage are found with development of CT.
血管密度高的核团包括了脑桥核、下橄榄核簇等与小脑相连系的核团，及楔束核、上橄榄核和展神经核；
The high density nuclei consist of the pontine nuclei, inferior olivary complex and so on which have connections with cerebellum, cuneate nucleus, superior olivary nucleus and abducent nucleus.
与脑桥诱导缓驰相关的抑制氨基酸释放的变化：在体微透析研究。
Changes in inhibitory amino acid release linked to pontine-induced atonia: an in vivo microdialysis study.
头部MRI检查可见左侧脑室旁、胼胝体梗死，右侧基底节、脑桥陈旧性腔隙性梗死；
Head MRI showed infarction of left paraventricular and corpus callosum, and old lacuna infarction of right basal ganglion and pons.
目的：提供有实用价值的小脑脑桥角手术的解剖学资料。
AIM: to provide the useful anatomic data for the microvascular surgery on cerebellopontine Angle.
目的探讨脑桥梗死的临床和影像学特点。
Objective To explore the clinical and imaging characteristics of pontine infarction.
实验结果表明：rtn和脑桥及延髓的呼吸相关结构之间存在纤维联系。
The above results show that the RTN has axonal connections with the pontine and medullary respiratory related structures.
探讨脑桥中央髓鞘溶解症的病因、临床特点、影像学表现、治疗及预后。
The pathogenic factors, clinic characteristics, iconography representation, treatment and sequel prophylaxis of central pontine myelinolysis were discussed.
探讨脑桥中央髓鞘溶解症的病因、临床特点、影像学表现、治疗及预后。
The pathogenic factors, clinic characteristics, iconography representation, treatment and sequel prophylaxis of central pontine myelinolysis were discussed.
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