• 方法晶体上皮细胞原代培养

    Methods: Bovine lens epithelial cells (BLEC) were cultured in vitro.

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  • 阴性对照组晶体上皮细胞阳性染色

    In negative control group rabbit LEC nuclei were not positive stained.

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  • 结论添加晶体皮质提高晶体上皮细胞培养成功率

    ConclusionAdding lens cortex can increase the success rate of primary culture of lens epithelial cells.

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  • 超微结构上,晶体上皮细胞晶体细胞出现明显病理改变。

    No obvious pathological lesions occurred in ultrastructure of lens epithelia and lens cells.

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  • 作用机制可能抑制晶体上皮细胞生长溶解纤维蛋白等

    The possible mechanism is the efficacy of heparin in inhibiting the growth of len epithelium, anti inflammatory and dissolving fibrin.

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  • 背景:研究发现晶体上皮细胞凋亡白内障发生共同细胞基础

    BACKGROUND: it was reported that the apoptosis of lens epithelia was the common cytological basis of the genesis of cataract.

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  • 其后混浊发生机制主要残留晶体上皮细胞迁移增殖转分化有关

    The mechanism of posterior capsule opacification is mainly related with migration, proliferation and transdifferentiation of remain crystal epithelial cells.

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  • 方法利用改良消化法晶体上皮细胞进原代培养培养的细胞进行形态学观察。

    Methods: Using the developed procedure to culture rabbit lens epithelial cells, and the cultured cells were examined morphologically.

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  • 结果显示人类晶体上皮细胞体外生存能力有限,体外生长能力供体年龄呈相关。

    The results revealed that the in vitro survival capacity of the human lens epithelial cells is limited and the proliferative capacity of the cells is negatively cor-related to the donor age.

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  • 方法:取新西兰白色家兔晶体上皮细胞进行原代培养细胞融合用胰蛋白酶进行消化传代

    Methods New Zealand white rabbit lens epithelial cells were primarily Cultured. After the confluence, the cells were trysinized and sub-cultured.

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  • 对照组晶体覆盖晶体上皮细胞,ASODN晶体后囊前无明显晶体上皮细胞增殖。

    The lens posterior capsule was covered by a layer of lens epithelium in the control group, while the lens epithelium could hardly been observed on the lens posterior capsule in the ASODN group.

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  • 对照组晶体覆盖晶体上皮细胞,ASODN晶体后囊前无明显晶体上皮细胞增殖。

    The lens posterior capsule was covered by a layer of lens epithelium in the control group, while the lens epithelium could hardly been observed on the lens posterior capsule in the ASODN group.

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