结论慢性间断性低氧能明显增加大鼠肝脏erd (CYP3A2)和ANH (CYP2E1)活性，其机制可能与其在转录水平上提高肝脏cyp3a2和CYP2E1的基因表达水平有关。

Conclusion Chronic intermittent hypoxia can increase rat liver microsomal ERD and ANH activities. The mechanism is related to up-regulation of CYP3A2 and CYP2E1 expression at the transcriptive levels.

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结论慢性间断性低氧能明显增加大鼠肝脏erd (CYP3A2)和ANH (CYP2E1)活性，其机制可能与其在转录水平上提高肝脏cyp3a2和CYP2E1的基因表达水平有关。

Conclusion Chronic intermittent hypoxia can increase rat liver microsomal ERD and ANH activities. The mechanism is related to up-regulation of CYP3A2 and CYP2E1 expression at the transcriptive levels.

youdao