致病性的朊病毒蛋白质在脑中沉积并损伤脑组织。
The malformed prion proteins can be deposited in the brain, thereby destroying brain tissue.
目的探讨损伤脑组织对骨髓基质细胞分化的诱导作用。
Objective To study the effect of damaged brain tissue on the differentiation of bone marrow stromal cells in rat.
目的研究脑醒喷鼻剂对大鼠局灶性脑缺血再灌注损伤脑组织超微结构的影响。
Objective To study the effects of Naoxing Nasal Spray(NNS)on ultrastructure of brain tissues in rats with focal cerebral ischemia -reperfusion injury(CIRI ).
目的研究雌激素对雄性大鼠脑缺血再灌注损伤脑组织兴奋性氨基酸(EAA)含量的影响。
Objective To study the effects of estrogen on the contents of EAA in cortex of male rats during cerebral ischemia and reperfusion.
这样高的含量表明，这些患病孩子的脑组织已经暴露在了高氧化压力下，有可能会造成累积的损伤。
Such high levels suggest the brains of autistic children are exposed to a lot of oxidative stress, something that would probably cause cumulative damage.
因此溶栓同时联合抑制炎症反应，可能对减轻缺血脑组织的损伤有益。
Therefore thrombolytic therapy combined with preventing inflammatory response might contribute to relieving cerebral tissue damage after ischemia.
结论：实验结果表明“冰黄液”对脑出血急性期大鼠脑组织损伤具有较好的保护作用。
Conclusion: Binghuang Liquid has a good protective effect on the injury of brain tissue of rat at acute term of cerebral hemorrhage.
结果：氟桂嗪治疗组在再灌注各个时间点脑组织损伤程度均明显轻于脑缺血组。
RESULTS: in flunarizine therapy group, the degree of injury of brain tissue was significantly lighter than that at each time in cerebral ischemia group.
目的：探讨脑表面降温对缺血再灌注损伤后的脑组织含水量和伊纹氏蓝含量的影响。
Objective:To investigate whether brain surface cooling(BSC) can decrease cerebral content of water and Evans Blue(EB) after ischemia-reperfusion injury in experimental rabbits.
结论血栓通能减轻脑组织的缺血再灌注损伤，改善神经功能缺失，其作用机制可能与增加HSP70表达有关。
Conclusion Xueshuantong can protect brain tissue from cerebral ischemia reperfusion injury and improve the nerve function. The mechanism may be related with the increasing of HSP70 expression.
结论：IL -8具有双重作用，既参与脑组织的正常代谢及生理功能，又参与了脑缺血损伤的病理过程。
Conclusion: IL 8 has double roles: it plays a role in metabolism and function in normal brain tissue and a role in pathological course of cerebral ischemia injury.
结果脑组织病理切片显示，模型组脑组织有严重损伤；
Results Severe damage to the brain tissue in the treated group was revealed by pathological biopsy.
结果迟发性颅内血肿、弥漫性脑肿胀、侧裂区脑挫裂伤、脑组织缺血、缺氧等是重型颅脑损伤术中急性脑膨出的主要原因。
Results The main cause of acute encephalocele were delayed intracranial hematomas, acute diffuse brain swelling, cerebral contusion and laceration in Sylvain tissue and ischemic anoxia.
血管性痴呆系由一系列脑血管病导致脑组织损伤所引起的痴呆综合征的总称。
Vascular dementia (VD) is the general term of dementia syndrome caused by the injury of brain tissue resulted from a series of cerebral vascular diseases.
资料提炼：共收集到50余篇关于2型受体生物学特性及其与组织损伤修复，特别是与心、肾、脑组织损伤修复相关的文章。
DATA EXTRACTION: a total of 50 papers about the bionomics of AT2 receptors and the renovation of damaged tissue especially on heart, kidney as well as renovation of brain tissue injury were collected.
方法建立大鼠急性脑缺血再灌流损伤模型，用原子分光光度仪检测脑组织电解质含量。
Methods Cerebral ischemia reperfusion injury model was produced in rats. The electrolyte contents were measured with atomic absorption spectrophotometer(AAS).
结论：脑梗死可致血清ace活性降低，与脑组织损伤程度有关，与脑功能状态有关。
CONCLUSION: the serum ACE activity decreases in the event of cerebral infarction, which is related with the degree of cerebral tissue damage and the state of brain function.
乙酰半胱氨酸组较模型组脑组织损伤轻，炎性细胞浸润少。
Compared with the model group, the injury of brain tissue was mild and inflammatory cells were fewer in the N-acetylcysteine group.
结论：大剂量纳络酮能够改善重症颅脑损伤引起的脑组织乏氧，改善患者预后。
Conclusion: Large dose of naloxone can improve the prognosis of severe head injury.
他解释说，血液中的这些化学物质在睡眠缺乏后上升，暗示缺觉会导致脑组织损伤。
The rise of the chemicals in the blood after sleep loss may suggest not getting enough sleep is conducive to a loss of brain tissue, he explained.
目的观察脑组织中血浆蛋白分布在损伤早期的改变。
Objective To observe the early changes of the distribution of plasma proteins in injured brain.
病理检查示no升高伴随着脑组织的损伤。
The histopathology showed that the increased NO was associated with the cerebral tissue damage.
本文主要综述炎症细胞、细胞因子、趋化因子、黏附分子等炎症因子对脑组织损伤的影响。
This paper would review the role of inflammatory cells and mediators such as cytokines, chemotactic factors, adhesion molecules on cerebral tissue injury.
目的观察早期环境干预对宫内感染致脑损伤仔鼠脑组织神经丝蛋白（NFP）的表达及对其神经行为的影响。
Objective To explore the effect of early enriched environment intervention on expression of neurofilament protein (NFP) in brain and the neurobehavior of filial rats with brain injury.
结论补体及炎症反应参与脑组织缺血再灌注损伤。
Conclusions Complement and inflammatory reaction participated in cerebral ischemia-reperfusion injury.
目的：研究褪黑素(MT)对谷氨酸所致脑组织氧化损伤模型小鼠的对抗作用。
OBJECTIVE: to study the counteraction of melatonin (MT) on the oxidative damage model of brain tissues of mice caused by glutamic acid.
目的：探讨氯化甲基汞(MMC)对发育脑组织损伤的机理。
Objective: To study the mechanism of Chloride methylmercury (MMC) damage of the developing brains.
目的建立人体正中矢状平面头部组织的平面应变模型，探讨脑组织变形与大脑损伤间的关系。
Objective To establish the plane strain model of brain tissues on mid-sagittal plane and discuss the relation between the brain tissue deformation and brain injury.
结果通脉胶囊可明显降低脑含水量、毛细血管通透性，减轻脑组织损伤。
The effects of Tongmai Capsule on water contents of brain, capillary permeability and injury of brain tissue were investigated.
目的研究氟中毒对脑组织的损伤，探讨氟中毒人群血清中脑髓鞘碱性蛋白(MBP)含量与脑髓鞘损伤的关系。
Objective To study the effect of fluorosis on brain, and correlation between serum MBP content and brain myelin sheath damage in endemic fluorosis areas.
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