能量耗竭引起损伤级联反应。
The energy failure leads to the development of a cascade of injury.
本文从脑缺血损伤级联反应及针刺结合药物的超早期(6小时内)治疗等方面进行了综述和探讨。
Thertefore, in the present paper cascade of damage and acupuncture treatment combined with medication at super early stage (within 6 hours) for cerebral ischemic injury, etc. were reviewed and...
本文从脑缺血损伤级联反应及针刺结合药物的超早期(6小时内)治疗等方面进行了综述和探讨。
Thertefore, in the present paper cascade of damage and acupuncture treatment combined with medication at super early stage (within 6 hours) for cerebral...
结论甘露醇可能通过影响炎症损伤级联反应的多个环节，抑制CEC、IL - 8等多种效应因子的产生和表达，从而对ALI发挥防治作用。
Conclusion Mannitol has a protective effect on rabbits with ALI through effecting various cascade reaction of inflammatory injury and down regulating of the generation and expression of CEC and IL-8.
通过动物实验，初步探讨IL-18在急性免疫性肝损伤中的意义，及早期使用IL-18抗体及时阻断细胞因子级联反应对保护肝组织的重要性。
To study rudimentarily the role of IL-18 in acute immunity hepatic injury, and the important of blocking cascade reaction of cytokines by exerting IL-18 antibody in early stage.
自由基在继发性损伤中的具体机制尚未完全清楚，可能与缺血级联反应、缺血再灌注和细胞凋亡有关。
The mechanisms contributing to the brain damage have not yet been well clarified. The mechanisms may be related with ischemic cascade reaction, ischemic reperfusion and cell apoptosis.
缺血性脑损伤是一个复杂的病理过程，提示只有联合使用作用于不同机制的脑保护剂，阻断其级联反应才可能取得更好疗效。
Ischemic brain damage is a complex process caused by mutiple pathological pathways, which indicates the joint use of the agents may block the cascade by different mechanisms and have better effects.
目的：评价大黄素抗脑缺血损伤作用，并从细胞因子水平及表达方面探讨其抑制炎性级联反应机制。
Objective: to assess emodin antagonism to cerebral ischemia injury, and to discuss the mechanism of emodin inhibiting the inflammatory cascade reaction from the levels and expressions of cytokines.
研究表明，其发病机理是由于一系列级联反应引起的神经元损伤和神经细胞凋亡。
Research indicated that the pathogenesis of AD is a series of cascades in brain inducing neuronal damage and neuronal apoptosis.
研究表明，其发病机理是由于一系列级联反应引起的神经元损伤和神经细胞凋亡。
Research indicated that the pathogenesis of AD is a series of cascades in brain inducing neuronal damage and neuronal apoptosis.
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