用声发射方法检测了复合材料损伤与断裂的全过程，证明了该结构的合理性与可行性。
The damage and fracture process of the composite are detected by acoustic emission. The experiment proves the simulating structure to be rational and feasible.
不锈钢长期在高温高压临氢环境下服役，其损伤与断裂对于石化企业的安全生产是至关重要的。
The damage and fracture of stainless steel serviced in hydrogen environment with high temperature and pressure are very important for the safety production of petroleum and chemical industries.
本论文立足于对混凝土材料特性的认识，提出一个进行混凝土损伤与断裂过程模拟的细观数值模型。
This paper is based on the understand of the concrete material. The mathematic model to study the process of concretes damage and fracture is being put forward.
过亚硝酸根对DNA的损伤主要有两种形式：dna碱基修饰与dna单链断裂。
Two forms of DNA damage caused by peroxynitrite are DNA base modification and DNA single strand breakage.
文中以裂纹连续扩展的断裂力学方法，研究单向纤维复合材料的细观损伤演化与韧度计算。
The methodology of fracture mechanics based on continuous crack growth was applied to the investigation of the microscopic damage evolution and toughness calculation for fiber composites.
准则表明最终的结构破裂取决于与临界损伤变量密切相关的断裂应变。
It is concluded that the final collapse of the structure is decided by the rupture strain that is deduced from the critical damage variable.
指出拉伸断裂是表面与内部损伤共同作用的结果。
It may be concluded that tensile fracture is caused by the combined effects of surface and internal damage.
计算了不同大地构造单元的断裂构造的分形维数，给出了其分形维数与损伤演化的关系。
The fractal dimension of the different geotectonic elements was calculated and the relation between the fractal distribution and the damage evolvement was given in this paper.
结论反式BPDE可导致DNA断裂损伤，其损伤程度与剂量有一定关系。
Conclusions Anti-BPDE can result in DNA strand breakage, and the degrees of damage is related to the administrated doses of anti BPDE.
氧化应激可以导致DNA链断裂、DNA位点突变、DNA双链畸变和原癌基因与肿瘤抑制基因突变等形式的DNA损伤；
Oxidative stress can cause DNA damage, such as DNA strand breaks, point mutations, aberrant DNA cross-linking, and mutations in proto-oncogenes and tumor suppressor genes;
氧化应激可以导致DNA链断裂、DNA位点突变、DNA双链畸变和原癌基因与肿瘤抑制基因突变等形式的DNA损伤；
Oxidative stress can cause DNA damage, such as DNA strand breaks, point mutations, aberrant DNA cross-linking, and mutations in proto-oncogenes and tumor suppressor genes;
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