• 目的观察复方刺蒺藜苷慢性轻度不可预见性应激抑郁模型大鼠行为学影响。

    Objective:To observe the effect of tribuloside on behavior and neurogenesis in dentate gyrus of chronic stress depression rats.

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  • 目的探讨电针会、太冲穴抑郁模型大鼠海马组织NGFBDNF影响。

    Objective:To investigate the effects of electroacupuncturing acupoints Baihui, Taichong on NGF, BDNFv of hippocampus of depression model rats.

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  • 目的观察脑缺血后抑郁模型大鼠下丘脑胺酸羟化酶(TH表达益肾调气方药干预作用。

    Objective:To observe the expression of Tyrosine Hydroxylase(TH) of Hypothalamus in post-stroke depression rat model and effects of Yishen Tiaoqi decoction.

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  • 五加胶囊可能通过改变抑郁模型大鼠结肠组织mda,SODCOX2表达来改善其结肠的功能。

    Ciwujia capsule can protect the colon tissue of depressed rats by changing the levels of MDA, SOD and the expression of COX 2.

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  • 目的建立利血抑郁模型合适剂量观察不同剂量利血平自发活动的影响。

    Objective: to establish the suitable dosage of reserpine for animal model of depression, and to observe the different dosage of reserpine on locomotion activity in rats.

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  • 方法建立经典大鼠抑郁模型

    Methods The classic depression model of rat was produced.

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  • 方法悬挂实验大鼠强迫游泳实验模型HPE的抗抑郁作用进行研究。

    Methods By using the mouse tail suspension test and rat forced swimming test models of depression, the antidepressant effects of HPE were observed.

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  • 方法采用慢性综合应激建立抑郁大鼠模型

    Methods Rat was treated with chronic and comprehensive stress to form depression model.

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  • 方法采用大鼠慢性应激抑郁模型、灌胃给药、行为学免疫组织化学方法。

    METHOD: This study adapted the depression rat model of chronic stress, behavior and immunohistochemistry.

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  • 建立抑郁模型

    The depression model of rats was produced.

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  • 目的观察针刺抑郁模型大鼠行为学前额皮质单胺类神经递质的影响分析针灸治疗抑郁可能作用机制

    AIM: To observe effects of acupuncture on praxiology and monoamine neurotransmitter in frontal cortex of depressed model rats and analyze the mechanism of acupuncture on treating depression.

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  • 目的研究抑郁模型大鼠腹腔注射5羟色氨酸(5 - HTP)引起海马CA 3区神经原放电变化

    Objective to explore the changes of neuron stimulation in the hippocampi CA3 region in the depression model rats after intraperitoneal injection of 5-htp.

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  • 采用养结合CUMS方式建立大鼠抑郁模型

    CUMS with solitary condition was taken to establish rat depression model.

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  • 应用分养长期不可预见性中等强度应激造成抑郁模型大鼠

    Separation feeding, long term unpredictability and medium stimulation stress techniques were applied to develop depression model rats.

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  • 目的观察菖欢胶囊慢性利血平孤养抑郁神经症模型大鼠行为影响

    Objective To investigate the effect of ChangHuan capsule on behavior in the separation and chronic reserpine-induced depressive neurosis rat model.

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  • 目的采用通量基因芯片观察大鼠抑郁模型基因表达变化结合基因功能分类体系探讨抑郁发病机制

    AIM: To observe the alteration of gene expression of depression rat models by gene chips of high flux, and explore the pathogenesis of depression combining with classification system of gene function.

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  • 模型制备:采用慢性不可预见性应激法造成大鼠抑郁模型

    Model preparation: The depression model rat was produced by chronic unpredictable stress.

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  • 方法:(1) 动物模型连续21慢性轻度不可预见性应激配合孤养方法建立抑郁模型

    METHODS: (1) The chronic stressed depression rats were established by chronic unpredictable mild stress and separation after 21 days.

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  • 目的:观察归对慢性不可预见性模型大鼠血清皮质酮雌激素(E2)水平影响,初步探讨其防治抑郁可能机制

    Objective: To study the effect of Guipi Decoction on depression model rats serum cortisol and estradiol (E2) and the mechanism is approached.

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  • 摘要目的建立阳虚抑郁大鼠病证结合动物模型进行评价

    Abstract: Aim To establish depression rat model with kidney-yang deficiency and evaluate the related parameters.

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  • 结论大鼠双侧动脉永久性结扎结合行为限制卒中后抑郁实验临床研究提供较为理想的动物模型

    Conclusion the permanent ligation of bilateral common carotid artery in combination of behavioral limitation could provide the ideal animal models for experiment and clinical study of PSD.

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  • 抑郁模型RA代谢信号通路失调。

    A dysregulated ra metabolism and signaling was also found in the hypothalamus of a rat model for depression.

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  • 慢性应激抑郁模型甘丙肽及其受体2部分区的高表达提示甘丙肽很有可能参与应激过程神经元功能调制

    The high expression of galanin and galanin receptor-2 in some brain area suggested that galanin probably take part in the modulation of the function of neurons during the stress process.

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  • 慢性应激抑郁模型甘丙肽及其受体2部分区的高表达提示甘丙肽很有可能参与应激过程神经元功能调制

    The high expression of galanin and galanin receptor-2 in some brain area suggested that galanin probably take part in the modulation of the function of neurons during the stress process.

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