在胚胎发育阶段，兴奋和抑制性神经元的适当比例确立以优化大脑功能。
During embryonic development, the proper ratio of excitatory and inhibitory neurons is established for optimal brain function.
首先，科学家发现唐氏综合症小鼠的胚胎中异常兴奋和抑制性神经元的比例异常，其中抑制性细胞的数量要高得多。
First, the scientists found abnormal proportions of excitatory and inhibitory neurons in the brains of the embryonic DS mice, with a much higher quantity of the inhibitory cells.
我们总结神经祖细胞和胶质祖细胞移植能够产生兴奋性和抑制性神经元，神经营养因子能够诱导移植物来源神经元轴突向指定方向生长。
We concluded that NRP/GRP grafts can be used to produce excitatory and inhibitory neurons, and neurotrophin gradients can guide axonal growth from graft-derived neurons toward putative targets.
再强调一遍,这些化学物质可使另一个神经元变得兴奋,提高该神经元产生神经冲动的可能性,也可抑制另一个神经的兴奋性
These--Again, the chemicals could excite the other neuron bring up the chances it will fire, or inhibit the other neuron .
结论：硝普钠可以抑制脊髓源性神经干细胞的增殖、诱导脊髓源性神经干细胞向神经元的分化。
Conclusions is Sodium nitroprusside can inhibit the proliferation and promote the differentiation of spinal neural stem cells into neuronal but not glial cells in vitro.
所有scn区域的神经元都可以产生这种抑制性神经递质，有实验证实每天早晨八点钟向SCN区细胞释放GABA能使它们同步化。
All SCN neurons make this inhibitory neurotransmitter, and it had been shown that giving GABA daily at 8 a. m. to SCN cells synchronizes them.
抑制性影响使下丘听神经元的频率调谐曲线变窄，听空间反应区域缩小，易化性影响与之相反。
Corticofugal inhibition sharpened the frequency tuning and reduced the auditory response area of IC neurons, and corticofugal facilitation produced the opposite effects.
结论2 ，5 己二酮可降低大鼠DRG感觉神经元和脊髓前角运动神经元（VSC4 。 1细胞）、内源性NGF的表达水平，并进一步抑制了两类细胞的生长与存活。
Conclusion The results suggested that 2,5-HD can decrease the endogenous NGF levels in both cultured DRG sensory neurons and VSC4.1 motor neuron cells of rats.
结论在慢性癫痫发作中，GABA能神经元的活性增强，考虑是由于癫痫过程中兴奋性增强，引起GABA能神经元抑制功能代偿性增加的结果。
There was significant difference between control and epileptic group. Conclusion The results suggest that the activity of GABA ergic neurons enhanced in chronic seizures.
结论异丙酚可能通过抑制大脑缺氧，抑制神经元的凋亡，从而对大鼠局灶性脑缺血-再灌注损伤发挥保护作用。
Conclusion Propofol may inhibit hypoxia in the brain and the apoptosis of nerve cells in result of protecting the cerebral ischemia and reperfusion damage in rats.
结论：尼莫地平可能通过抑制HO - 1表达、维持铁离子稳态，减轻铝过负荷所致的神经元退行性变。
CONCLUSION: Nimodipine relieves the neurodegeneration induced by aluminum overload through inhibiting the expression of HO-1 and keeping the homeostasis of iron.
本文主要介绍高级中枢、缺氧后脑血流量变化、缺氧对呼吸中枢神经元的实质性损害、神经递质和神经调质在缺氧性呼吸抑制过程中的作用。
This article introduces the effect of high central nervous system, changes of cerebral blood flow after hypoxia, damage of hypoxia to neurons in the respiratory center, neurotransmitters a…
传入性肌肉内神经节突触前抑制作用的减弱影响了那些支配拮抗肌传入性的中间神经元对拮抗肌的抑制作用，导致了这种互相抑制作用的衰减。
This decreased reciprocal inhibition is due to reduced presynaptic inhibition of the muscle afferent input onto the inhibitory Ia interneuron innervating the muscle antagonist to the input.
传入性肌肉内神经节突触前抑制作用的减弱影响了那些支配拮抗肌传入性的中间神经元对拮抗肌的抑制作用，导致了这种互相抑制作用的衰减。
This decreased reciprocal inhibition is due to reduced presynaptic inhibition of the muscle afferent input onto the inhibitory Ia interneuron innervating the muscle antagonist to the input.
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