建立COP-1免疫的慢性高眼压大鼠模型。
To establish reliable copolymer-1(COP-1)-immunized chronic EIOP rats models.
观测大鼠正常眼压值，建立稳定的大鼠慢性高眼压模型。
To detect the normal intraocular pressure (IOP) and establish reliable chronic elevated intraocular pressure (EIOP) rat models.
目的探讨针刺对家兔慢性高眼压及慢性高眼压下视网膜超微结构的影响。
OBJECTIVE To investigate effect of acupuncture on experimental chronic ocular hypertension and retinal ultrastructure in rabbits.
方法于兔眼前房注射1%甲基纤维素每周1次，连续5周，制作慢性高眼压模型。
Methods Rabbit chronic ocular hypertension model was Established by injecting 1% methylcellulose into anterior chamber once a week for 5 weeks.
巩膜表浅静脉烧灼法制成慢性高眼压模型，分别于实验后1和2个月观察视网膜损害情况。
The tissue damage with chronic hypertension, induced by the cauterization of two episcleral veins, was studied at 1 and 2 month after the treatment.
目的：探讨银杏叶提取物联合灯盏细辛对兔慢性高眼压模型视网膜神经节细胞的保护作用。
AIM: to study the protective effect of Ginaton and fleabane on retinal ganglion cells in rabbit chronic high intraocular pressure model.
藏红花提取液在高眼压早期能有效提高慢性高眼压下视网膜中SOD活性，降低MDA含量。
Crocus sativus can effectively enhance the activity of SOD and reduce the content of MDA in retina in early period of OHT.
目的探讨慢性高眼压后兔眼视神经轴突数目的变化情况及藏红花提取液对视神经轴突损伤的保护作用。
Objective To explore the number change of optic nerve axons in rabbits with chronic ocular hypertension (OHT) and the protective effect of crocus sativus extract on the optic nerve axons injury.
诱导后晚期的模型眼前房角胶原增生，结构破坏。结论复方卡波姆诱导的兔眼慢性高眼压模型房水流出受阻的主要部位在小梁内皮网部。
Conclusions the obstruction of aqueous humor outflow induced by compound Carbomer in rabbit high IOP model is caused mainly by the changes in trabecular endothelial cells.
诱导后晚期的模型眼前房角胶原增生，结构破坏。结论复方卡波姆诱导的兔眼慢性高眼压模型房水流出受阻的主要部位在小梁内皮网部。
Conclusions the obstruction of aqueous humor outflow induced by compound Carbomer in rabbit high IOP model is caused mainly by the changes in trabecular endothelial cells.
应用推荐