• 结论彩色多普移植慢性排斥反应作出评价。

    Conclusion: the color doppler echocardiography can be used to assess the chronic renal transplantation rejection.

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  • 慢性排斥反应有肺泡上皮细胞增生纤维结统组织增生。

    The chronic rejection manifested epithelioid cells of lung alveoli and interstitial fibrous tissue proliferation.

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  • 目的探讨HCMV移植慢性排斥反应作用机制

    Objective: to analysis the role of human cytomegalovirus (HCMV) infection in the pathogenesis of chronic liver rejection.

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  • 目的探讨小剂量FK778大鼠移植慢性排斥反应的预防作用。

    Objective To study the effect of low-dose of FK778 in preventing chronic renal allo-graft rejection in rats.

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  • 结论H BO预防减轻同种移植肾慢性排斥反应中能重要作用

    Conclusions HBO could play important role in preventing or reducing chronic allograft rejection.

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  • 目的探讨肝脏移植远期以黄疸特征慢性排斥反应的发生原因应对策略。

    Objective To explore the cause and treatment strategy of chronic graft rejection characteristic of jaundice in patients with liver transplantation.

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  • 目的探讨慢性排斥反应移植动脉硬化增殖细胞核抗原(PCNA)表达关系

    Objective To investigated the relationship between the expression of PCNA and transplant arteriosclerosis of chronic rejection.

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  • 活检病理结果慢性排斥反应2例,胆汁淤积性肝硬化1例,肝脏局灶性坏死1例。

    The results of biopsy showed 2 chronic rejections, 1 biliary stasis hepatic cirrhosis and 1 hepatic focal necrosis.

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  • 心脏移植后的急性慢性排斥反应一直阻碍心脏移植广泛应用重要因素。

    But the graft rejection, both acute and chronic rejection, is still an obstacle for heart transplantation.

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  • 结果48标本中,有15例提示有急性排斥反应12提示慢性排斥反应

    RESULTS. : Among 48 cases, 15 cases showed ACR and 12 showed CR in at least one organ.

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  • 方法应用显微外科技术制作移植慢性排斥反应模型受体大鼠随机分为

    Methods The rat model of chronic renal allograft rejection was made with micro surgery. The recipients were divided into two groups.

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  • 探讨慢性排斥治疗方法对59移植术后发生慢性排斥反应患者的治疗情况进行总结

    To investigate the treatment of chronic rejection, 59 cases of chronic rejection (CR) following renal allograft were clinically summarized.

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  • 目的研究血管细胞粘附分子-1(VCAM-1)心脏移植慢性排斥反应中的表达及作用。

    Objective: To investigate the expression of the vascular cell adhesion molecule-1(VCAM-1) in allograft chronic rejection(CR) after heart transplantation in rats.

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  • 结果表明ICAM-1VCAM-1正常肾脏慢性排斥反应移植肾脏上表达分布不同

    The results showed that there were different distribution of ICAM 1 and VCAM 1 expression in nomal kidney and renal allograft during chronic rejection.

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  • 结论OKT3治疗难治性排斥反应效果优于ATG,尤其对再次移植患者慢性排斥反应患者无效。

    Conclusion OKT3 is better than ATG for reversing the refractory rejection, especially in retransplant patients, but it has no any effect on chronic rejection.

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  • 目前移植领域一个重要方向就是诱导多机制起作用免疫耐受,防治人体同种异体器官的慢性排斥反应

    At present, the immunological tolerance in transplantation induced by multi-mechanisms, to inhibit the chronic graft rejection, represents an important trend in the field of transplantation.

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  • 这项研究回顾了数年前接受治疗患者研究者能够找到发生慢性排斥反应患者同那些发生慢性排斥反应患者之间差异

    Because this study looked back at patients treated years ago, the researchers could look for differences between patients who subsequently suffered chronic lung rejection and those who did not.

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  • 结论门静脉注射两种以上供体细胞减轻慢性排斥反应VEGFET-1表达血管硬化及纤维化程度呈正相关。

    Conclusions Injection of two or more kinds of donor live cells may alleviate chronic rejection via decreasing of expressions of VEGF and ET-1 in the grafts.

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  • 目的探讨干预慢性迟发性超反应(DTH)CD 8 +T淋巴细胞细胞毒等效应机制同种小鼠心脏移植后慢性排斥反应影响

    Objective to evaluate the effect of delayed type hypersensitivity (DTH) and CD8 t lymphocyte cytotoxicity intervention on the development of chronic rejection in mouse cardiac allografts.

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  • 有效地控制慢性移植排斥反应诱导移植免疫耐受当今移植免疫学研究热点

    The control of chronic transplant rejection and the induction of transplant graft tolerance today are the hottest research field in transplant immunology.

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  • 方法随机观察移植患者41例其中出现排斥反应16(急性排斥12例,慢性排斥4例)。

    Methods Randomly investigate 41 liver transplantation patients, among them there were 16 patients with reject reaction (including 12 with acute rejection, 4 with chronic rejection).

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  • 高倍镜下,慢性血管排斥反应引起肾动脉显著增厚纤维化

    At high magnification, the renal arteries with chronic vascular rejection are markedly thickened and fibrotic.

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  • 高倍镜下,慢性血管排斥反应引起肾动脉显著增厚纤维化

    At high magnification, the renal arteries with chronic vascular rejection are markedly thickened and fibrotic.

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