而大多数现有的针对恶性胶质瘤的临床治疗方式只作用于一条通路。
Most standard clinical treatments for glioblastoma currently target just one pathway.
目的探讨脑恶性胶质瘤长期生存患者的主要治疗方法和安全性。
Objective to evaluate the main treatment methods and safety for long-time survival patients of glioblastoma.
目前，胶质瘤特别是恶性胶质瘤的治疗还是一个非常棘手的问题。
At present time, the therapy of glioblastoma, especially the malignant glioblastoma is very difficulty.
恶性胶质瘤是脑癌中最致命的类型，目前对此尚无有效的治疗方法。
Malignant glioma is among the deadliest types of brain cancer for which there currently is no effective treatment.
恶性胶质瘤是侵袭程度最高的脑瘤之一，病人存活时间基本不超过两年。
Glioblastoma is one of the most aggressive brain tumors, killing nearly every patient within two years.
它们组成了几乎百分之八十的癌细胞生长，就像肯尼迪议员的恶性胶质瘤。
What they make up almost eighty percent of cancer growths, like the malignant glioma thats Senator Kennedy has.
到目前为止，20位患有恶性胶质瘤的脑瘤患者中有12位对这种治疗反应良好。
So far, 12 of 20 patients with a type of brain tumor called glioblastoma have responded well to the treatment.
背景与目的：探讨NDV(新城鸡瘟病毒)修饰的瘤苗治疗脑恶性胶质瘤的疗效。
BACKGROUND AND AIM: to evaluate the therapeutical effect of autologous Newcastle_Disease_Virus (NDV) modified tumor cell vaccine in the treatment of malignant gliomas.
该受体在介导人恶性胶质瘤细胞的迁移、存活、生长及血管发生中起重要作用。
The receptor has important function on mediating migration, survival, growth and vasculogenesis of the human malignant glioma cells.
目的通过对比观察脑恶性胶质瘤术后替莫唑胺胶囊联合外放射治疗的疗效及安全性。
Objective To compare the curative effects and safety of temozolomide plus radiotherapy in the treatment of post-operative malignant glioma.
恶性胶质瘤是所有脑瘤中最为常见和致命的一种，预计它每年影响着全球24万人。
Glioblastomas, the most common and lethal of all brain cancers, affect an estimated 240,000 people around the world per year.
目的观察在三维胶原体外培养条件下恶性胶质瘤细胞体外诱导内皮细胞血管生成的作用。
Objective To investigate the effects of malignant glioma cells on the induction of endothelial cells to form tubule-like structure in a three dimensional culture system with collagen in vitro.
干细胞具有向恶性胶质瘤趋向性迁移的特性，并可能成为恶性胶质瘤基因治疗的理想载体。
Various studies have demonstrated the tremendous tropism of stem cells for malignant gliomas, making these cells a potential vehicle for delivery of therapeutic genes to disseminated glioma cells.
SWI序列显示的高度恶性胶质瘤组的出血率大于低度恶性胶质瘤组，但没有统计学意义；
There was no statistical significance in the incidence of blood products detected on SWI between low potential malignancy and high potential malignancy groups;
结论结合CT表现与病人年龄、病变部位，全面分析，能提高大脑恶性胶质瘤的诊断准确率。
Conclusion the correct diagnosis of brain malignant glioma could be established by comprehensive consideration of the ct appearance, the patient's age and the location of tumor.
目的探讨正电子发射断层扫描（PET）在恶性胶质瘤治疗后疗效观察及预后评价中的作用。
Objective To investigate the value of positron emission tomography (PET) in testing the treatment efficacy and the role of predictive evaluation in malignant gliomas.
研究人员发现，在实验室关闭RAD51分子，杀死恶性胶质瘤细胞，提高了放射治疗的效果。
Researchers discovered that switching off the RAD51 molecule increased the effectiveness of radiotherapy to kill glioblastoma cells in the lab.
研究人员已经建议一些特殊种类的恶性胶质瘤细胞可以繁殖，复制出相同的拷贝从而更加的耐治疗。
Researchers had suggested that some specialised groups of glioblastoma cells can reproduce to make identical copies that are more resistant to treatment.
目的探讨热休克蛋白90 (HSP90)在人脑恶性胶质瘤的表达及其在肿瘤增殖、分化中的作用。
Objective To probe the expression and role of the heat-shock protein 90 (HSP90) in the proliferation and differentiation of human brain gliomas.
约2%至3%受到辐射的雄鼠患上了恶性胶质瘤——它是一种脑部癌症——而没有受到辐射的对照组没有此类病例。
About 2 to 3 percent of the male rats exposed to the radiation developed malignant gliomas, a brain cancer, compared with none in a control group that was not exposed to radiation.
最常见的类型的脑癌即恶性胶质瘤以出现类似癌干细胞的脑细胞为特征，这些细胞不是替代损伤细胞，而是形成肿瘤。
The most common type of brain cancer-glioblastoma-is marked by the presence of these stem-cell-like brain cells, which, instead of triggering the replacement of damaged cells, form cancer tissue.
埃伯哈特和他的同事们研究了恶性胶质瘤的细胞系以发现γ-内分泌酶抑制剂的影响，这种药物可阻断促使肿瘤增长的Notch受体途径。
Eberhart and colleagues studied glioblastoma cell lines to investigate the effects of a gamma-secretase inhibitor, a medication that blocks the Notch receptor, on tumor growth.
更深入的研究将会阐释恶性胶质瘤中NotchHedgehog和Wnt通路之间的关系，并寻找使肿瘤产生耐药性的其他信号传导途径，他说。
Further studies will examine the relationship among the Notch, Hedgehog and Wnt pathways in glioblastoma and look for other signaling processes that help tumors become resistant to therapy, he says.
由一家美国公司Introgen 设计的Advexin目标在于攻克头颈部癌症；位于伦敦的ArkTherapeutics生产出的Cerepro将目标对准了恶性胶质瘤，一种致死性的脑肿瘤。
Advexin, devised by Introgen, an American company, is aimed at combating head and neck cancer; and London-based Ark Therapeutics’ Cerepro targets malignant glioma, a fatal brain tumour.
他的医生认为根源在于恶性神经胶质瘤，即脑部肿瘤，并预测病情不容乐观。
His doctors determined the cause was a malignant glioma, a brain tumor that carries a grim prognosis.
这些基因改变是胶质瘤恶性生物学行为的基础，但对此的了解还不是很清楚。
Such gene expression changes are the molecular basis of biological behaviors of malignant glioblastoma, but still not well known.
目的：研究LRRC4基因在恶性脑胶质瘤细胞系中的表达情况。
Objective To examine the expression absence of LRRC4 gene in glioblastoma cell lines.
目的：研究LRRC4基因在恶性脑胶质瘤细胞系中的表达情况。
Objective To examine the expression absence of LRRC4 gene in glioblastoma cell lines.
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