拷贝数分析显示肿瘤具有异质性，具有异源性分化的恶性叶状肿瘤有不同的肿瘤演化。
Copy number analysis revealed intratumoral heterogeneity and evidence for divergent tumor evolution in malignant phyllodes tumors with and without heterologous differentiation.
本研究结果表明，新的通路参与恶性叶状肿瘤的发病机制，显著提高了我们对肿瘤生物学的认识，具有潜在的临床意义。
The results identify novel pathways involved in the pathogenesis of malignant phyllodes tumors, which significantly increase our understanding of tumor biology and have potential clinical impact.
我们测序了10例恶性叶状肿瘤的510个癌症相关基因，其中5例伴有脂肪肉瘤样分化，1例伴有粘液软骨肉瘤样分化。
We sequenced 510 cancer-related genes in 10 malignant phyllodes tumors, including 5 tumors with liposarcomatous differentiation and 1 with myxoid chondrosarcoma-like differentiation.
我们测序了10例恶性叶状肿瘤的510个癌症相关基因，其中5例伴有脂肪肉瘤样分化，1例伴有粘液软骨肉瘤样分化。
We sequenced 510 cancer-related genes in 10 malignant phyllodes tumors, including 5 tumors with liposarcomatous differentiation and 1 with myxoid chondrosarcoma-like differentiation.
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