比较骨形态发生蛋白2 (BMP - 2)基因治疗与生长因子缓释方法修复节段性骨缺损效果。
To compare the effects between gene therapy and gradual release carrier for bone morphogenetic protein-2 (BMP-2) in repairing bone defects.
研究表明，骨形态发生蛋白-2（BMP-2）与骨组织肿瘤之间存在高度相关性。
Previous studies have indicated the close relation between bone tumors and bone morphogenetic protein-2(BMP-2).
目的探讨带血供肌瓣作为骨形态发生蛋白(BMP)载体修复骨缺损的可行性。
Objective to investigate the effect of vascular muscle flap as a carrier of bone morphogenetic protein (BMP) to repair long bone defect.
两组在骨折后3周和6周的骨形态发生蛋白2及血管内皮生长因子差异无显著性意义（P > 0.05）。
There was no significant difference of BMP-2 and VEGF between experimental group and control group at 3 weeks and 6 weeks after fracture (P > 0.05).
由于其与骨形态发生蛋白1 (BMP - 1)在结构和功能上具有高度的同源性，因而也属于BMP - 1类分子。
This protein is highly homologous to morphogenetic protein-1 (BMP-1) in terms of both structure and function, thus belonging to BMP-like protease.
由于其与骨形态发生蛋白1 (BMP - 1)在结构和功能上具有高度的同源性，因而也属于BMP - 1类分子。
This protein is highly homologous to morphogenetic protein-1 (BMP-1) in terms of both structure and function, thus belonging to BMP-like protease.
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