结论急性播散性脑脊髓炎的MRI表现有定特征性，MRI具有重要诊断价值。
Conclusion ADEM carries certain characteristics on MRI. MRI is of great value in its diagnosis.
目的探讨实验性自身免疫性脑脊髓炎(eae)大鼠脑血管内皮细胞表面分子的改变。
Objective to explore the surface molecule changes of the experimental autoimmune encephalomyelitis (EAE) rat brain vascular endothelial cells.
目的：用髓鞘碱性蛋白(MBP)诱导实验性变态反应性脑脊髓炎(eae)豚鼠模型。
Objective: To induce the EAE model on Guinea pig wi th Myelin Basic Protein (MBP).
应用1，25二羟基维生素D3治疗和预防实验性变态反应性脑脊髓炎，可使其症状缓解。
The application of 1, 25-dihydroxyvitamin D3 can treat and prevent EAE and made the its symptom relieve.
目的建立实验性变应性脑脊髓炎(eae)动物模型，为进一步研究其发病机制提供依据。
Objective To construct animal model for further elucidating the pathogenesis of experimental allergic encephalomyelitis (EAE).
目的：研究DT390-IL2重组质粒对实验性变态反应性脑脊髓炎（EAE）的治疗效果。
Objective:To study the curative effect of EAE with DT390-IL2 recombinant plasmid.
前言：目的：研究实验性变态反应性脑脊髓炎(eae)大鼠外周免疫器官中细胞凋亡的情况。
Objective: to investigate the apoptotic cell in the peripheral lymphoid organs in experimental autoimmune encephalomyelitis (EAE) rats.
目的：用全脊髓匀浆（粗制髓鞘碱性蛋白）建立豚鼠实验性自身免疫性脑脊髓炎（EAE）模型。
Objective:To establish the experimental autoimmune encephalomyelitis(EAE) model on Guinea Pig with pan-spinal cord homogenate(coarse myelin basic protein, cMBP).
一氧化氮在多发性硬化的经典动物模型—实验性变态反应性脑脊髓炎的预防和治疗中发挥重要作用。
Nitric oxide plays an important role in the prevention and treatment of experimental allergic encephalomyelitis, which is the classical experimental model of multiple sclerosis.
方法符合临床诊断标准的急性播散性脑脊髓炎患者21例，18例发病前有病毒感染或疫苗接种史。
Methods 21 cases of clinically proven ADEM were retrospectively reviewed. 18 patients had a history of viral infection or vaccination prior to the onset of ADEM.
目的探讨儿童急性播散性脑脊髓炎（ADEM）的临床经过及随访结果，以期在诊治水平上有所提高。
Objective To explore the clinical course and follow-up of acute disseminated encephalomyelitis(ADEM)in children and improve the ability of diagnosis and therapy.
目的探讨实验性自身免疫性脑脊髓炎（EAE）中枢神经系统（ CNS）中浸润细胞的凋亡情况。
Objective To study the apoptotic cells in central nervous system (CNS) with experimental autoimmune encephalomyelitis (EAE) at various stages of the course.
科尔博士因此指出肌痛性脑脊髓炎可能有其生物学根源，而且使人们有理由希望，验血可以帮助识别不同的类型。
This, says Dr Kerr, points to a biological basis for the illness and holds out hope that a blood test could be developed to identify its different forms.
方法通过对13例临床诊断为急性播散性脑脊髓炎的患儿进行回顾性分析，探讨该病的临床特点、治疗及预后。
Methods a retrospective analyses and study up on 13 children with the clinical diagnosis of ADEM to identify the clinical features, treatment and prognosis.
实验性自身免疫性脑脊髓炎(eae)是一种免疫细胞介导的以中枢神经系统白质损坏为特征的自身免疫性疾病。
Experimental allergic encephalomyelitis (EAE) is an immune disease, whose character is the damage of white matter of nervous system mediated by immunocyte.
研究者在每个大鼠品系建立实验性自身免疫性脑脊髓炎动物模型，并检测疾病进展过程中这些动物都有哪些变化。
The researchers established EAE in each mouse strain and examined what was common to all of the animals when they developed disease.
目的探讨实验性自身免疫性脑脊髓炎(eae)动物模型血cd4 +CD 25 +T细胞的变化及其意义。
Objective to study the number changes of blood CD4 + CD25 + t cells and its significance in rats with experimental autoimmune encephalomyelitis (EAE).
目的：探讨血管细胞粘附分子- 1 (VCAM - 1)在实验性自身反应性脑脊髓炎(eae)致病中的作用。
Objective: to explore the role of vascular cell adhesion molecule-1 (VCAM-1) in the pathogenesis of experimental autoimmune encephalomyelitis (EAE).
目的：观察实验性变态反应性脑脊髓炎模型大鼠外周血cd4 +，CD 8 +的改变及免疫调节剂左旋咪唑的作用。
AIM: To observe the changes of CD4 +, CD8 + and effect of levamisole in experimental allergic encephalomyelitis rats.
目的建立实验性自身免疫性脑脊髓炎(eae)动物模型，研究腹腔内注射可溶性髓鞘碱性蛋白(MBP)诱导免疫耐受的作用机制。
Objective to establish the animal model of EAE (experimental autoimmune encephalomyelitis) to study the inducible suppression of EAE by i. p. administration of soluble MBP (myelin basic protein).
同时他还说：“这个研究结果强调了这样一个事实——血脑屏障的通透性改变是发生象实验性变应性脑脊髓炎(一种多发性硬化病的动物模型)这类中枢神经系统感染性疾病的重要因素。”
"This puts an emphasis on the fact that blood brain permeability changes are an important aspect of the development of a CNS inflammatory disease like EAE, an animal model of MS," he says.
同时他还说：“这个研究结果强调了这样一个事实——血脑屏障的通透性改变是发生象实验性变应性脑脊髓炎(一种多发性硬化病的动物模型)这类中枢神经系统感染性疾病的重要因素。”
"This puts an emphasis on the fact that blood brain permeability changes are an important aspect of the development of a CNS inflammatory disease like EAE, an animal model of MS," he says.
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