小脑性共济失调患者可出现轮替运动障碍。
Individuals with cerebellar ataxia could display dysdiadochokinesia.
最主要的临床表现为眩晕、小脑性共济失调。
The typical clinical manifestations were vertigo and cerebellar ataxia.
目的：探索线粒体dna点突变与遗传性共济失调的关系。
Objective: to study the possible relationship between mitochondrial DNA point mutations and hereditary ataxia.
方法对36例急性共济失调患儿的临床资料进行回顾性分析。
Methods The clinical datas of 36 infant patients suffering from acute ataxia are analyzed retrospectively.
目的探讨感觉性共济失调型CIDP的临床特点和发病机理。
Objective To explore the clinical characteristics and pathogenic mechanism of sensory ataxia form of GBS.
目的研究线粒体dna点突变与遗传性共济失调(HA)的关系。
Objective to study the possible relationship between mitochondrial DNA point mutations and hereditary ataxia (HA).
目的研究遗传性脊髓小脑性共济失调7型（SCA7）的基因突变和临床特征。
Objective To study the gene mutation and clinical characteristic of hereditary spinocerebellar ataxia type 7 (SCA7).
目的研究细胞凋亡在脊髓小脑性共济失调3型(SCA3)分子发病机制中的作用。
Objective to study the role of cell apoptosis in the molecular pathogenesis of spinocerebellar ataxia type 3 (SCA3).
回顾小脑性共济失调的研究历史、分类，介绍脊髓小脑共济失调的病因机制和治疗方法。
The history of the research of cerebellar ataxia, and the different classifications of ataxia were reviewed. The pathogenesis and treatments of spinocerebellar ataxia were summarizd.
目的总结小儿急性共济失调的常见类型与病因方法对36例急性共济失调患儿的临床资料进行回顾性分析。
Objective To summarize the common types and medical reasons for acute infant ataxia. Methods The clinical datas of36infant patients suffering from acute ataxia are analyzed retrospectively.
目的分析泛素依赖的蛋白水解通路(UPP)在转染了脊髓小脑性共济失调3型(SCA3)基因的PC 12细胞中的定位及作用。
Objective To analyze the role and location of ubiquitin-dependent proteolysis pathway (UPP) in PC12 cells transfected by plasmids with spinocerebellar ataxia type 3 (SCA3) gene.
结论纯运动性偏瘫患者，如果出现构音障碍、头晕及共济失调时应考虑到桥脑梗死的可能。
Conclusion The patients with pure motor hemiparesis and dizziness, ataxia, dysarthria have more possibility to suffer from pontine infarction.
入院时，患者定向力正常，但表现出严重的构音障碍，左侧中枢性面瘫，共济失调步态。
On admission, he was oriented, but had severe dysarthria, left-sided central facial palsy and gait ataxia.
入院时，患者定向力正常，但表现出严重的构音障碍，左侧中枢性面瘫，共济失调步态。
On admission, he was oriented, but had severe dysarthria, left-sided central facial palsy and gait ataxia.
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