目的：化疗药物对急性髓性白血病细胞体外毒性进行相关性研究。
Objective:To study the correlation between cytotoxicity of daunorubicine (DNR) and harringtonine (H) to leukemic cells in vitro from 39 patients with acute myeloblastic leukemia (AML).
疾病可引发脊髓发育不良、脊髓增殖失调，急性髓性白血病和支气管瘤。
Conditions included myelodysplastic syndrome, myeloproliferative disorders, acute myeloid leukemia, and bronchial carcinoma.
在第一方面的实施方案中，所述急性髓性白血病是复发性急性髓性白血病。
In an embodiment of the first aspect, the acute myelogenous leukemia is relapsed acute myelogenous leukemia.
存在甲基化的急性髓性白血病病人外周血标本中，RIZ1基因表达缺失或降低。
The expression of RIZ1 mRNA was found to be lower in the acute myeloid leukemia samples with RIZ1 promoter methylation.
MA9常见于急性髓性白血病，偶尔也在急性淋巴白血病中发现，并且会导致较差的预后。
MA9 is commonly found in acute myeloid leukemia (AML) and occasionally in acute lymphoid leukemia and is associated with intermediate to poor outcome.
研究方法：用流式细胞仪检测30例急性髓性白血病的P_(170)和活化t细胞的表达。
Methods: Flow cytometer (FCM) was used in detection of the expression of P170 and active t cell in leukemic cells from 30 cases of AML patients.
目的探讨70岁以上老年急性髓性白血病临床及生物学特征、影响预后的因素及个体化治疗方案。
Objective to explore the clinical and biological features, prognostic factors and individualized treatment regimen of acute myelogenous leukemia patients over 70 years old.
目的：研究昆布多糖硫酸酯（LAMS）对急性髓性白血病HL - 6 0细胞株的调控作用。
Objective:To study the effects of LAMS on modulation of HL-60 cell strain.
结果：急性髓性白血病病人标本与正常人标本比较，RIZ1基因的表达下降，差异具有统计学意义（P<0.05）。
Results: Compared with normal person, the RIZ1 gene expression level decreased significantly in acute myeloid leukemia(P<0.05).
给华盛顿大学捐赠细胞的该名女性患有急性髓性白血病，这是一种进展非常快的癌症，美国每年有13000人患病，其中8800人死亡。
The woman at Washington University had acute myelogenous leukemia, a fast-growing cancer that affects about 13,000 people a year in the United States and kills 8,800.
我们进行了一项临床试验旨在分析伏立康唑在急性髓细胞白血病化疗诱导阶段预防肺部浸润的有效性和安全性。
We conducted a trial to analyze the efficacy and safety of voriconazole in the prevention of lung infiltrates during induction chemotherapy for acute myelogenous leukaemia (AML).
在急性髓细胞性白血病aml和急性淋巴细胞性白血病all中，原始的急性白血病细胞进展、形成多达上亿个的白血病细胞。
In acute myelogenous leukemia (AML) and acute lymphocytic leukemia (ALL), the original acute leukemia cell goes on to form about a trillion more leukemia cells.
探讨老年急性髓细胞性白血病的有效治疗方案。
To explore of the efficient treating method for acute myeloid leukemia in the elderly.
目的报道1例以CD7+和双重t（8；21）易位为特征的近四倍体克隆急性髓细胞性白血病（AML）。
Objective To report a case of acute myeloid leukemia (AML) with CD7 + near-tetraploid clones by doublet(8; 21).
MDS是一组异质性很强的疾病，其特征为：血细胞减少、无效造血及进展为急性髓系白血病风险。
MDS is a group of very heterogeneous disorders characterized by cytopenias, inefficient hematopoiesis and the risk of progression into acute myeloid leukemia.
“慢性”白血病少有或几乎没有母细胞。与急性白血病相比，“慢性淋巴细胞性白血病”和“慢性髓细胞性白血病”通常进程相对缓慢。
"Chronic" leukemias have few or no blast cells. "chronic lymphocytic leukemia" and "chronic myelogenous leukemia" usually progress slowly compared to acute leukemias.
急性髓细胞性白血病(aml)很难治愈，不过可喜的是近年来疗效已有所改善。
AML is a hard blood cancer to cure. The good news is that there has been improvement in treatment results in recent years.
【译】结合在细胞遗传学正常的急性髓细胞性白血病的分子和复发和生存的预后指标。
Combined Molecular and Clinical Prognostic Index for Relapse and Survival in Cytogenetically Normal Acute Myeloid Leukemia.
摘要：老年急性髓系白血病(aml)患者治疗耐受性差，预后不佳。
ABSTRACT: Elderly patients with acute myeloid leukemia (AML) tolerate intensive chemotherapy poorly and usually have poor prognosis.
预激方案；急性髓细胞性白血病；粒细胞集落刺激因子；
Priming induction regimen; Acute myeloid leukemia(AML); Granulocyte colony stimulating factor;
作者建议治疗有血液和骨髓癌症的病人，或者急性前髓球性白血病。
The authors recommended that the treatment be given to patients with blood and bone marrow cancer, or acute promyelocytic Leukemia.
与急性髓细胞性白血病相比，急性淋巴细胞性白血病细胞免疫功能更为低下。
Compared with acute myelogenous leukemia, patients with acute lymphoblastic leukemia had more poor cellular immune function.
结论：DCIK能诱导机体产生特异性的免疫反应，对急性髓细胞性白血病的治疗有较好的临床疗效。
Conclusion: DCIK can induce specific immunoreaction in the immune system and has satisfactory clinical anticancer efficacy in treatment of AML.
目的观察131I-GMCSF诱导HL60/ADM细胞凋亡，探讨其诱导HL60/ADM细胞凋亡的机制，比较131I-GMCSF对HL60/ADM细胞和HL60细胞的细胞毒作用，为其临床治疗难治性或复发性急性髓细胞性白血病提供实验依据。
Objective: To compare the cytotoxicity of HL60/ADM cells and HL60 cells treated with 131I-GMCSF and to investigate the mechanism of the apoptosis of HL-60/ADM cells induced by 131I-GMCSF.
目的观察131I-GMCSF诱导HL60/ADM细胞凋亡，探讨其诱导HL60/ADM细胞凋亡的机制，比较131I-GMCSF对HL60/ADM细胞和HL60细胞的细胞毒作用，为其临床治疗难治性或复发性急性髓细胞性白血病提供实验依据。
Objective: To compare the cytotoxicity of HL60/ADM cells and HL60 cells treated with 131I-GMCSF and to investigate the mechanism of the apoptosis of HL-60/ADM cells induced by 131I-GMCSF.
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