目的观察中药复方温心汤对家兔心肌缺血再灌注损伤心肌细胞凋亡以及病理组织学改变的影响。
Objective To observe mainly the effect of the Chinese traditional medicine (compound Wenxin soup) on myocyte apoptosis and path-histology change in rabbits ischemia referpusion injury.
目的：研究组织因子（TF）反义寡脱氧核苷酸（AS/TF）对大鼠心肌缺血再灌注损伤的作用。
Objective To study the effects of antisense oligodeoxynucleotide(AS/TF) for rat tissue factor (TF) on the myocardial ischemic reperfusion injury.
结论MG- 132通过降低缺血再灌注心肌组织中炎症因子的表达，抑制了再灌注损伤。
Conclusion Myocardial reperfusion injury can be inhibited by proteasome inhibitor MG-132 through suppressing the expression of inflammatory factor in rats.
目的探讨大鼠心肌缺血及再灌注过程中血清白介素10 (IL 10)、心肌组织中髓过氧化物酶(mpo)活性的变化和相互关系。
Objective To investigate the changes of interleukin 10 (IL 10) and MPO and their relation in acute myocardial ischemia and reperfusion.
超声组织定征定量分析缺血再灌注损伤心肌。
Myocardial I-R injury was quantitive analysed by ultrasound tissue characterization.
再灌注损伤可能与再灌注过程中心肌组织内源性儿茶酚胺大量释放有关。
These studies suggest that the postischemic reperfusion injury may be, in part, due to endogenous catecholamine release during reperfusion.
测定缺血前、再灌注后心肌组织SOD含量，心肌收缩功能，观察各组心肌hsp72表达。
The SOD content in myocardial tissue, myocardial systolic function were detected before ischemia and after reperfusion, and the myocardial HSP72 expressions in all the groups were observed.
结论在大鼠心肌缺血再灌注损伤中心肌组织的损伤程度与心肌组织中浸润的中性粒细胞有密切相关性。
CONCLUSON the myocardium injury correlates with the neutrophils infiltrated in the myocardium on myocardium ischemic reperfusion in rat.
目的：应用定量组织速度成像技术（QTVI）测定急性前壁心肌梗死（MI）后不同时段左室的收缩功能，评价再灌注治疗对急性前壁MI患者左室收缩功能的短期影响。
Objective: To assess the short period effects on the systolic function of left ventricular of reperfusion therapy in acute myocardial infarction (AMI) by quantitative tissue velocity imaging (QTVI).
无再流现象是指冠状动脉闭塞，血流中断后重新恢复血流，却无心肌组织的有效灌注的现象。
No reflow phenomenon refers to non-effective perfusion in myocardium when the flow regains after flow blockage resulting from coronary artery occlusion.
结论：大鼠心肌组织存在钙敏感受体，其表达与月龄有一定关系，并可能参与心肌缺氧-再灌注损伤的发生。
CONCLUSION: Our results demonstrate that the CaSR is expressed in the rat cardiac tissue. The mRNA expression of CaSR changes with age and is involved in the anoxia-reperfusion injury.
本文以组织化学方法探讨家兔心肌缺血不同时间后再灌注对琥珀酸脱氢酶(SDH)活性的影响。
By histochemical means, we observed the effects of reperfusion on succinate dehydrogenase (SDH) activity after acute myocardial ischemic injury in rabbits.
本文以组织化学方法探讨家兔心肌缺血不同时间后再灌注对琥珀酸脱氢酶(SDH)活性的影响。
By histochemical means, we observed the effects of reperfusion on succinate dehydrogenase (SDH) activity after acute myocardial ischemic injury in rabbits.
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