充血性心力衰竭所致的心房间质纤维化，通过影响心房传导来促进心律失常的发生。
Congestie heart failure produces atrial interstitial fibrosis, which promotes arrhythmogenesis by interfering with atrial conduction properties.
结果窦性心律时窦房结发出冲动，激动右心房上部并向右心房下部和左心房传导，而房颤时双心房均可见方向不一、片段的激动波或大的折返环，可见多个传导阻滞区。
During acute AF, there are multiple and discrete excitation wave fronts or reentry activities were found in both LA and RA, and bidirectional block regions could be shown.
结果显示：特发性房颤患者的心房有效不应期缩短，相对不应期延长，早搏刺激的房间传导延缓显著增加。
The results showed that patients with IAF had shorter atrial effective refractory periods and longer atrial relative refractory periods, and also longer interatrial conduction delays.
对心房和房室传导系统ERP亦有延长作用。
It also prolonged atrial ERP and the ERP of atrioventricular conduction system.
结论：心脏起搏抑制房颤发生的机制可能与快频率起搏加速心房内传导、抑制了房性期前收缩、消除了窦性心动过缓和心脏长短间歇有关。
Conclusion: atrial overdrive can prevent AF, which may results from accelerating interatrial conduction, inhibiting atrial premature beat, eliminating sinus bradycardia and long short interval.
它是目前直接了解窦房结的功能、窦房结与心房之间传导情况的最佳方法。
Hence SNE_b is the best non-invasive method available for examination of sinus node function and sino-atrial conduction.
尤其在窦房结和心房之间的传导组织有时发生故障时这点尤为重要。
This is particularly important when the conducting tissues between the SA node and the ventricles fail only from time to time.
目的了解食道心房调搏在房室传导功能检测中的意义。
Objective To understand the value of the transesophageal pacing in test of atrio-ventricular conduction function.
方法对39例PR间期延长(研究组)和141例PR间期正常(对照组)患儿房室传导功能进行了经食道心房调搏研究。
Methods The transesophageal pacing was performed in 39 children with prolonged pr interval (study group) and 141 children with normal pr interval (control group).
并对随着基础频率心房起搏的增加房室结呈递减性传导是频率滤过作用的基础等生理现象做出解释。
With the foundation and increase the frequency of atrial pacing was reduced atrioventricular node conduction is based on the frequency filtering effect of other physiological phenomena to explain.
结论心房起搏频率的改变可影响房室结的有效不应期和传导功能。
Conclusion the atrial pacing rate of change can affect the AV node effective refractory period and conduction in refractory period.
瘢痕组织通过阻断紊乱的电传导而终止房颤，该紊乱的电传导会使心房不规律收缩。
The scar tissue stops atrial fibrillation by interfering with chaotic electrical signals that cause the atria to contract irregularly.
瘢痕组织通过阻断紊乱的电传导而终止房颤，该紊乱的电传导会使心房不规律收缩。
The scar tissue stops atrial fibrillation by interfering with chaotic electrical signals that cause the atria to contract irregularly.
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