结论海马神经元突触结构异常是低压低氧条件下大鼠学习记忆脑高级功能障碍的形态学基础。
Conclusions the structural abnormalities of the hippocampal neuronal synapses is the morphological basis of abnormalities of higher brain functions of learning and memory induced by hypobaric hypoxia.
大脑组织这种形态结构的改变可能导致其神经元功能障碍，进而引起运动性中枢疲劳的发生。
The change of ultra-structure in cerebral cortex microcirculation can induce the dysfunction of cholinergic neuron and further cause the exercise-induced central fatigue.
血管内皮功能障碍是动脉粥样硬化的最敏感指标之一，它出现于血管形态学改变之前。
Vascular endothelial dysfunction is one of the most sensitive indicators for atherosclerosis, which displays prior to the changes of vascular morphology.
方法应用组织病理学半定量方法观察了86例临床无肾功能障碍的老年人尸检肾小球的形态学改变。
Methods Histopathologically semiquantitative method was used to study renal glomeruli in 86 aged autopsied patients without clinical renal dysfunction.
方法应用组织病理学半定量方法观察了86例临床无肾功能障碍的老年人尸检肾小球的形态学改变。
Methods Histopathologically semiquantitative method was used to study renal glomeruli in 86 aged autopsied patients without clinical renal dysfunction.
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