心肌氧化应激和硫醇敏感的信号转导分子参与心肌肥厚和纤维化的发病过程。
Myocardial oxidative stress and thiol-sensitive signaling molecules are implicated in pathogenesis of hypertrophy and fibrosis.
建立小鼠成纤维细胞系nih - 3t3应激适应细胞模型，为应激适应研究提供平行可比的实验对象。
Establish stress adaptation model of mouse fibroblast cell line NIH-3T3, and provide a group of parallel object for stress adaptation research.
目的：探讨烧伤后慢性应激大鼠海马神经元微管相关蛋白- 2 (MAP - 2)和神经胶质细胞胶质纤维酸性蛋白(GFAP)表达的变化。
Objective: To study the expressions of microtubule associated protein-2 (MAP-2) and glial fibrillary acidic protein (GFAP) in hippocampus of rats with depression after burn.
睾酮；氧化应激；衰老；心肌成纤维细胞；表型。
Testosterone; Oxidative stress; Senescence; Cardiac fibroblasts; Phenotype.
内质网应激很可能是IETM促进肝纤维化乃至肝硬化发生的重要机制。
Endoplasmic reticulum stress is a possible mechanism in the development of liver cirrhosis promoted by IETM.
结论：黄芪注射液可以减弱CP的氧化应激，抑制PSC活化，缓解胰腺的纤维化。
Conclution: Radix Astragali injection can decrease the effect of oxidative stress of CP and inhibit activation of PSC, resulting in alleviating the fibrosis of the pancreas.
结论：黄芪注射液可以减弱CP的氧化应激，抑制PSC活化，缓解胰腺的纤维化。
Conclution: Radix Astragali injection can decrease the effect of oxidative stress of CP and inhibit activation of PSC, resulting in alleviating the fibrosis of the pancreas.
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