结论:趋化因子CXCL12、受体CXCR4和CXCL12—CXCR4生物学轴在肿瘤转移中发挥着重要的作用。
Conclusion: The chemokine factor CXCL12, its receptor CXCR4 and biological axis of CXCL12-CXCR4 play important roles in tumor metastasis.
它们的这些生物学功能是通过配体与受体结合的方式来发挥的,而CXCR3与CCR5是RA中趋化因子的标志性受体。
All of these biological effects are performed by binding of ligands to receptors. Elsewhere, CXCR3 and CCR5 are mark chemokine receptors in RA.
它们的这些生物学功能是通过配体与受体结合的方式来发挥的,而CXCR3与CCR5是RA中趋化因子的标志性受体。
All of these biological effects are performed by binding of ligands to receptors. Elsewhere, CXCR3 and CCR5 are mark chemokine receptors in RA.
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