• 目的研究麦芽醇大鼠脑缺血灌注损伤保护作用

    Objective to investigate the protective effects of maltol (MT) on cerebral ischemia and reperfusion damages in rats.

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  • 目的探讨辛伐他汀抗鼠脑缺血再灌注损伤的作用机制

    Objective To investigate the effects of simvastatin on cerebral injury induced by ischemia reperfusion in rats and the mechanisms.

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  • 目的研究利多卡因复方甘油大鼠脑缺血再灌注后脑水肿影响。

    AIM: To study the effect of lidocaine and compound glycerin on cerebral edema of rats after reperfusion.

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  • 结论老龄大鼠脑缺血再灌注脑组织损伤超载自由基损伤有关。

    Conclusion: the calcium overload and free radical injury were correlated with brain ischemia reperfusion injury in the aged rats.

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  • 目的探讨人工合成e -选择素鼠脑缺血再灌注损伤保护作用机制

    Aim: To investigate the mechanism of the protect role induced by preventive application of artificial synthesized E-selectin in cerebral ischemia-reperfusion injury of rat.

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  • 目的观察黄芩苷鼠脑缺血再灌注损伤保护作用初步探讨作用机制

    Objective To explore the protective effect of baicalin on the cerebral ischemia-reperfusion injury in rats and its mechanism.

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  • 目的探讨脂肪组织来源神经干细胞移植治疗大鼠脑缺血灌注损伤疗效

    Objective To study the effects of neural stem cells from human adipose tissue-derived stromal cells transplantation for treatment of cerebral ischemic - reperfusion injury in rats.

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  • 结论:微粉水蛭可以明显改善鼠脑缺血再灌注损伤而且作用效果明显优于粉。

    Conclusion: hirudo could improve the ischemia-reperfusion injury in rats, and it excels coarse smashing hirudo in effect.

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  • 目的探讨胸段膜外阻滞HTEA大鼠脑缺血再灌注损伤作用可能机制

    Objective To investigate the effects of high thoracic epidural anesthesia (HTEA) on cerebral ischemia-reperfusion (I/R) injury, and the possible mechanism.

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  • 结论青年老龄鼠脑缺血再灌注脑组织损伤NO含量降低TNF增高有关。

    Conclusions the brain ischemia reperfusion injury in young and the aged rats were correlated with the decrease of NO level and the increase of TNF.

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  • 注射AQP4基因升高大鼠脑缺血再灌注损伤早期脑内aqp4表达水平。

    Expression of AQP4 in the brain at the early period of cerebral ischemia reperfusion injury was increased after injection of APQ4 gene into the rat brain.

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  • 目的研究复方阿魏实验性鼠脑缺血再灌注损伤保护作用探讨作用机制

    OBJECTIVE: to study the protection of compound ferulic acid against cerebral ischemic reperfusion injury in experimental rats and to explore its function mechanism.

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  • 结论低温减轻大鼠脑缺血灌注损伤,降低脑组织HSP70GFAP蛋白表达

    Conclusion Mild hypothermia therapy may inhibit the expression of HSP70 and GFAP after cerebral ischemia and reperfusion injury in rats.

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  • 目的研究雌激素雄性鼠脑缺血灌注损伤脑组织兴奋氨基酸(EAA)含量影响

    Objective To study the effects of estrogen on the contents of EAA in cortex of male rats during cerebral ischemia and reperfusion.

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  • 方法采用局灶缺血模型,观察辛伐他汀鼠脑缺血再灌注损伤作用及钙蛋白酶活性变化。

    Methods The model of focal cerebral ischemia reperfusion in rat was established. The activity of calpain was measured dynamically by biochemical methods.

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  • 结论具有明显改善大鼠脑缺血再灌注损伤作用机制血清sodMDA变化有关

    Conclusion the eye acupuncture therapy played a role in improving cerebral ischemia-reperfusion injury evidently and the mechanism was related to the alteration of serum SOD and MDA levels.

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  • 前言:目的:观察大鼠脑缺血再灌注V CAM 1表达白细胞血管内皮细胞粘附变化

    Aim: To investigate the expression of VCAM-1 and changes of adhesion between leukocytes and endothelial cells in focal cerebral ischemic reperfusion rats.

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  • 结论丹皮酚可能具有抑制鼠脑缺血再灌注ICAM -1蛋白表达的作用,从而减轻了神经元损伤

    CONCLUSION: Paeonol may inhibit ICAM 1 protein expression after focal cerebral ischemia reperfusion in rats so as to relieve neuron damage.

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  • 目的探讨大鼠脑缺血再灌注损伤麻黄碱治疗星形胶质细胞胶质原纤维酸性蛋白(GFAP)表达水平影响

    Objective To investigate the effects of ephedrine on glial fibrillary acidic protein (GFAP) expression after cerebral ischemia-reperfusion injury in rats.

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  • 目的探讨鼠脑缺血再灌注缺血不同时相转录修复偶联因子CSB/ERCC6 )表达神经元DNA损伤的作用。

    Objective To investigate the transcription-coupled repair factor CSB/ERCC6 expression and neuronal DNA damage in ischemic area after cerebral ischemia-reperfusion in rats.

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  • 目的通过比较鼠脑缺血再灌注损伤基质金属蛋白酶- 9 (MMP - 9)表达,研究中药制剂通心络缺血脑组织的作用机制。

    ObjectiveTo investigate the effect of Tongxinluo by the dynamic changes of the expression of matrix metalloproteinase-9 (MMP-9) on local cerebral ischemic-reperfusion injury in rats.

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  • 目的探讨大鼠脑缺血再灌注损伤生长相关蛋白- 43 (GAP - 43)胰岛素样生长因子- 1 (IGF - 1)表达

    Objective to explore the expressions of growth associated protein-43 (GAP-43) and insulin-like growth factor-1 (IGF-1) in nervous system after cerebral ischemia reperfusion injury in rats.

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  • 方法采用血管闭塞法制备鼠脑缺血灌注模型后,二苯胺测定脑缺血再灌注金纳多治疗组随缺血再灌注时间延长皮质DNA裂解变化

    Methods The rats cerebral ischemia and reperfusion model by four -vessel occlusion method was used to observe the change of DNA splitting rate between ischemia and reperfusion group and Ginaton group.

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  • 课题利用大鼠脑缺血灌注模型,观察人参缺血再灌注后脑组织损伤影响,目的是探讨人参总皂甙缺血再灌注脑组织保护作用机制。

    In our study we investigated the influence of ginseng total saponin on brain tissue injury after ischemia-reperfusion by the model of brain ischemia-reperfusion with rat models.

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  • 目的观察美满霉素鼠脑缺血灌注后脑组织基质金属蛋白酶-2(MMP-2)基质金属蛋白酶-9(MMP-9)表达影响,探讨其神经保护机制。

    Objective To observe the effects of minocycline (MIN) on expression of MMP-2 and MMP-9 in Wistar rats with focal cerebral ischemic reperfusion.

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  • 结果黄芩苷可以明显改善大鼠脑缺血再灌注所致行为学障碍降低梗死,降低脑组织含水量,同时可以降低NO、NOSMDA的含量,增加SOD含量。

    The effects of baicalin on ischemia-induced barriers to behavior, rate of infarction, water content of brain tissue, contents of NO, NOS, MDA and SOD in brain tissues were observed.

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  • 目的观察强度半导体激光(LISCL)疗法对缺血灌注il-6影响

    Objective to observe the influence of low intensity semiconductor laser (LISCLT) on the IL - 6 in cerebral ischemia and reperfusion rats.

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  • 目的观察皂素局灶性脑缺血再灌注损伤保护作用

    Objective: To observe the protective effect of tea saponin on Focal cerebral ischemia-reperfusion injury.

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  • 目的观察皂素局灶性脑缺血再灌注损伤保护作用

    Objective: To observe the protective effect of tea saponin on Focal cerebral ischemia-reperfusion injury.

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