目的研究N-去硫酸肝素对大鼠肝缺血再灌注损伤的防治作用。
To investigate the effects of N - desulfated heparin on liver ischemia and reperfusion injury.
目的探讨大鼠缺血再灌注肝一氧化氮合酶的变化及其意义。
Objective To study the level of nitric oxide synthase(NOS)in ischemia-reperfusion rat liver.
前期的药理试验证明，其水提物对大鼠实验性肝缺血再灌注损伤模型(IR)有明显的保肝作用，而且进一步的研究表明主要起作用的成分为核苷类。
The primary experiment has revealed that its aqueous extract has obvious effects of liver protection based on IR model, and that the main functional components are nucleosides.
方法：测定大鼠肝脏局部缺血再灌注损伤和应用山莨菪碱时肝组织PAF含量、丙二醛（MDA）含量及肝脏病理变化。
Methods:Tissue PAF and malondialdehyde (MDA) contents were measured in rats with or without anisodamine treatment during hepatic ischemiareperfusion injury.
方法：测定大鼠肝脏局部缺血再灌注损伤和应用山莨菪碱时肝组织PAF含量、丙二醛（MDA）含量及肝脏病理变化。
Methods:Tissue PAF and malondialdehyde (MDA) contents were measured in rats with or without anisodamine treatment during hepatic ischemiareperfusion injury.
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