目的：探讨激活素a在新生大鼠缺氧缺血性脑损伤中的表达规律及其意义。
Objective To investigate the effects of EGB on hepatic fibrosis and expression of Activin a in rats with fibrosis.
结论：大鼠尾加压素II是大鼠肺主动脉有效的血管收缩剂，细胞信号转导通路MAPK的激活参与其收缩效应。
CONCLUSION: rat U-II was a potent vasoconstrictor of rat main pulmonary arteries and this response was mediated through MAPK.
结论：大鼠尾加压素II是大鼠肺主动脉有效的血管收缩剂，细胞信号转导通路MAPK的激活参与其收缩效应。
CONCLUSION: rat U-II was a potent vasoconstrictor of rat main pulmonary arteries and this response was mediated through MAPK.
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