结论：雷公藤红素对TNBS诱导的大鼠结肠炎具有显著保护作用，抑制促炎细胞因子的产生可能是其主要作用机制之一。
Conclusions: Tripterine has noticeable protective effects on TNBS-induced colitis in rats, suppression of proinflammatory cytokines may be one of its main mechanisms.
目的探讨凝血酶对体外培养的原代大鼠脑微血管内皮细胞(bmvec)的损伤作用及凝血酶抑制剂水蛭素的保护作用。
AIM to investigate the apoptotic effect of thrombin on brain microvascular endothelial cell (BMVEC) of cultured rat, and the protective effect of hirudin, an inhibitor of thrombin.
结论：辛夷挥发油可能通过抑制糖尿病大鼠血清及肾组织中P选择素蛋白表达发挥对肾脏的保护作用。
Conclusion: VOMBP can protect the kidney in rats with diabetic nephropathy by inhibiting the expressions of Pselectin protein in serum and in renal tissue.
促红细胞生成素与一氧化氮合酶抑制剂对肝硬化大鼠高动力循环状态的影响。
Effects of erythropoietin or nitric oxide synthesis inhibitor on hyperdynamic circulatory state in cirrhotic rats.
结论心理应激可不同程度地抑制大鼠腺垂体促甲状腺素细胞合成和分泌促甲状腺素。
Conclusion the synthesis and secretion of thyrotrophin of pituitary in rats may be inhibited by psychological stress.
结论：心肌肽素通过抑制心肌细胞凋亡，减轻心肌细胞结构损伤发挥对大鼠心肌缺血再灌注损伤的保护作用。
CONCLUSION: Cardiomyopeptidin plays a role in protecting myocardial ischemia-reperfusion injury in rats by inhibiting myocardial apoptosis and alleviating myocardial structural lesions.
ACN对大鼠睾丸中ABP和抑制素基因表达水平有影响，提示丙烯腈对睾丸中支持细胞可能有损伤作用。
The results indicate that ACN could induce changes in the levels of ABP and inhibin gene expression in testes of male SD rats, that may damage Sertoli cells in testes.
目的探讨凋亡基因对大鼠心肌梗死的表达及血管紧张素转化酶抑制剂(acei)的干预作用。
Objective to investigate the expression of apoptosis - related genes in rats with myocardial infarction and effects of ACEI.
结论：灯盏花素对糖尿病大鼠肝、肾组织有明显保护作用，其机制部分可能与抑制肝、肾组织氧化应激增加有关。
CONCLUSION: the mechanism of protective effect of breviscapine on liver and kidney may be at least partly correlated with the suppression of increase in oxidative stress in diabetic rats.
结果：不同川芎显著地抑制去甲肾上腺素所致大鼠胸主动脉条收缩，显著地抑制离体兔耳灌流量。
Methods: By the use of rat isolated aortic strips and rabbit isolated perfused intact ears, we observed the effects of different Chuanxiongs on the contraction of vascular smooth muscles.
目的：观察米诺环素对内毒素诱导的大鼠眼内炎症的抑制作用。
AIM: to investigate the inhibitory effect of minocycline on the ocular inflammation induced by lipopolysaccharide in the rats.
结论胰岛素对大鼠再灌注心肌损伤具有保护作用，其保护机制可能与抑制细胞凋亡及抗氧自由基作用有关。
Conclusion Insulin has a protective effect against myocardial ischemia and reperfusion injury in rat, the protective mechanism involves antiapoptotic and antioxide free radical effect of insulin.
结论：葛根素对d -半乳糖诱导的大鼠糖基化反应具有明显的抑制作用。
Conclusion: Puerariae can significantly inhibit glycation reaction in rats induced by D-galactose.
采用新生大鼠胰岛离体培养方法，观察了五肽胃泌素（G-5）对IL-1B致胰岛B细胞分泌抑制的影响。
The effect of pentagastrin (G-5) on interleukin-1B(IL-lB) inhibited insulin secretion in culturednewborn rat islet B-cells were observed.
目的观察血管紧张素转化酶抑制药卡托普利对大鼠局灶性脑缺血再灌注损伤后炎症反应的影响。
Purpose to investigate the anti-inflammatory effect of angiotensin-converting enzyme inhibitor captopril on the brain against ischemia-reperfusion injury and explore its mechanism of action.
目的观察血管紧张素转换酶抑制剂苯那普利减轻幼龄肾病大鼠蛋白尿的作用。
Objective To observe the effect of benazepril, an angiotensin converting enzyme inhibitor (ACEI) , on reducing proteinuria in a nephrotic young rat model of with massive proteinuria.
结论：芝麻素能纠正高脂、高糖诱导大鼠糖脂代谢异常，并抑制血压升高、改善血管重构。
CONCLUSION: Sesamin ameliorates disorders of glucose and lipid metabolism and inhibits vascular remodeling in rats caused by chronic high-fat, refined-sugar diet.
当Sink在维克森林大学的同事发现药物可以保护大鼠免受辐射造成的脑损伤后，她决定研究有中枢作用的血管紧张素转换酶抑制剂类药物对痴呆症风险的影响。
Sink decided to investigate the effect of centrally acting ACE inhibitors on dementia risk after her Wake Forest colleagues found the drugs protected rats from brain injury due to radiation.
结论脂糖舒对2型糖尿病模型大鼠不仅有明显降糖降脂、改善胰岛素抵抗和胰岛素敏感性作用，而且对其血浆蛋白非酶糖化和脂质过氧化亦有显著的抑制作用。
Conclusion ZTS has remarkable effect of lowering the insulin resistance and inhibiting the non-enzyme glycosylation of serum protein and the lipid-peroxidation in type-2-diabetes rats.
结论脂糖舒对2型糖尿病模型大鼠不仅有明显降糖降脂、改善胰岛素抵抗和胰岛素敏感性作用，而且对其血浆蛋白非酶糖化和脂质过氧化亦有显著的抑制作用。
Conclusion ZTS has remarkable effect of lowering the insulin resistance and inhibiting the non-enzyme glycosylation of serum protein and the lipid-peroxidation in type-2-diabetes rats.
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