目的探讨补体系统固有成分C9及补体调节因子CD59在大鼠急性脊髓损伤组织中的表达。
Objective To discuss the expression of an inherent component of complement system C9 and a complement regulatory factor CD59 in rat acute spinal cord injury tissue.
结论甲基强的松龙、MK- 801及两药联合对大鼠急性脊髓损伤的治疗作用在目前的实验条件下无明显差异。
Conclusion the administration of methylprednisolone or MK-801 alone or in combination has not different efficacy obviously existing experiment condition for the treatment of acute spinal cord injury.
目的研究预防使用大剂量甲基强的松龙对急性脊髓损伤大鼠的神经功能保护作用。
Objective To study the neuroprotective effect of high-dose methylprednisolone in prophylaxis in rat acute spinal cord injury.
目的：探讨大剂量甲基强的松龙对急性脊髓损伤大鼠神经功能的预防保护作用。
Objective: To study the preventive effect of high-dose methylprednisolone on acute spinal cord injury in rats.
现将主要结果简述如下：1。行为学改善方面：我们着手研究电针治疗急性脊髓损伤模型大鼠的机制。
Main results are sketched now as follows: 1. Behavior learns to improve aspect: We set about the mechanism studying the electricity needle cures the acute spinal cord injury model rat's.
能减轻大鼠中度急性脊髓损伤后的神经元和神经胶质细胞凋亡。
It can decrease the apoptotic neurons and gliocytes of the SD rats after moderate contusive SCI.
能减轻大鼠中度急性脊髓损伤后的神经元和神经胶质细胞凋亡。
It can decrease the apoptotic neurons and gliocytes of the SD rats after moderate contusive SCI.
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