单纯缺血组永久性结扎成年大鼠双侧颈总动脉建立大鼠cvi模型。
The CVI models were produced by permanent ligation of bilateral common carotid arteries.
实验研究部分采用了反复夹闭大鼠双侧颈总动脉配合硝普钠降压法复制SD大鼠拟血管性痴呆模型。
The vascular dementia model of SD rat was duplicated by clamping bilateral arteria carotis communis repeatedly and depressurization with Sodium Nitroprusside.
结论大鼠双侧颈总动脉永久性结扎结合行为限制可为卒中后抑郁的实验及临床研究提供较为理想的动物模型。
Conclusion the permanent ligation of bilateral common carotid artery in combination of behavioral limitation could provide the ideal animal models for experiment and clinical study of PSD.
目的观察舒降之对双侧颈总动脉重度狭窄的血管性认知障碍大鼠模型认知功能障碍的影响。
Objective To establish the rat models with severe carotid artery stenosis and to observe the effect of Statins on the spatial memory.
方法通过结扎双侧颈总动脉造成脑缺血模型，测定小鼠小肠的推进率及大鼠的胃酸分泌、胃蛋白酶的活性。
Methods the model was established by bilateral general cervical artery ligation mice intestinal motion rate, peptic acid secretion and the activity of pepsin were detected.
方法采用反复夹闭双侧颈总动脉伴低血压造成拟VD大鼠模型，通过原位末端标记（TU NEL）法检测脑神经细胞凋亡情况。
Method Experimental VD model rats were caused by clipping two side of total artery in neck repeatedly and to examine cerebral neurocyte apoptosis with TUNEL.
方法：采用动脉夹箝闭双侧颈总动脉的方法，制作脑缺血再灌注大鼠模型。
Methods: Rat model of cerebullar ischemia-and-reperfusion was made by using bulldog clamp incarcerating common carotid artery.
结果双侧颈总动脉结扎后小鼠的肠蠕动减慢，大鼠胃酸分泌减少，胃蛋白酶活性减弱。
Results The intestinal peristalsis moved slow, peptic acid secretion decreased and pepsin activity decreased.
结果双侧颈总动脉结扎后小鼠的肠蠕动减慢，大鼠胃酸分泌减少，胃蛋白酶活性减弱。
Results The intestinal peristalsis moved slow, peptic acid secretion decreased and pepsin activity decreased.
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