结论低温对大鼠内毒素性ARDS有一定的有利影响。
Conclusion Hypothermia had favorable effect on ARDS induced by lipopolysaccharide in rats.
方法首先使用埃氏大肠杆菌死菌液制备大鼠内毒素性休克模型；
Methods Endotoxic shock in rats was induced by intravenous injection of dead gramegative bacteria E.
为了进一步支持我们的假设，我们还采用大鼠内毒素耐受模型进行了研究。
In order to further support our hypothesis, endotoxin tolerance model has been used in our studies.
目的：观察人血浆高密度脂蛋白(HDL)对大鼠内毒素血症的治疗和防护效果。
AIM: to observe the therapeutic and protective effects of high-density lipoprotein (HDL) of human plasma on rat endotoxemia.
用大肠杆菌内毒素（LPS）复制大鼠内毒素性休克模型，氨基胍治疗组采用氨基胍治疗。
Rats were challenged by E. coli LPS to set up the model of endotoxic shock, AG group were treated by aminoguanidine.
本研究观察大鼠内毒素血症时肺组织中及外周血多形核中性粒细胞（PMN）凋亡、坏死及功能改变的差异。
To study the difference of changes on apoptosis, necrosis and respiratory burst of the polymorphonuclear neutrophils (PMN) in endotoxemia rat model.
结论肝硬化大鼠内毒素和细菌转位可能是由于IBO和肠通透性增加的结果，而IBO的发生可能是由于肠转运时间延长所致。
Conclusion Endotoxin and bacterial translocation in cirrhotic rats may be the result of IBO and higher permeability. IBO may be the result of longer transit.
用鲎试剂法测定用药后大鼠和人体血浆内毒素的含量变化。
Endotoxin concentrations in the plasma of rats and human body were measured through limulus test.
目的探讨利多卡因及山莨菪碱对急性坏死性胰腺炎（ANP）大鼠血淀粉酶、磷脂酶A2、内毒素及其病理学变化的影响。
Objective To investigate the effect of lidocaine and anisodamine on the alternations of serum amylase, phospholipase A2(PLA2), endotoxin and pathology in acute necrotizing pancreatitis(ANP) rats.
目的探讨内毒素预处理对大鼠前脑缺血再灌注后海马神经元的保护效应及其可能机制。
Objective To investigate the protective effect of endotoxin pretreatment on hippocampal neurons in rat forebrain following ischemia reperfusion and its possible mechanism.
目的：研究大黄对内毒素性休克大鼠炎性介质作用的机制。
Objective: to study the mechanism underlying rhubarb inflammatory mediator response in endotoxic shock.
方法：采用大肠杆菌内毒素复制大鼠内毒性热瘀证模型基础上，检测桃核承气汤对血液流变学和凝血指标的影响。
Method: Take Bacillus Coli endotoxin to copy rats endotoxic heat - stasis syndrome model, then test the effect of PRC on rheology and coagulation indexes.
结论：大鼠饥饿后早期确有肠黏膜组织结构受损，发生内毒素移位，同时伴有肠黏膜免疫学屏障受损。
CONCLUSION: the structure of intestinal mucosa is damaged in the earlier stage of starvation in rats, accompanied by endotoxin translocation and dysfunction of intestinal mucosal immune barrier.
目的：研究钙增敏剂mci - 154对内毒素休克大鼠心肌钙稳态的影响。
Objective: to investigate the effects of MCI 154, a calcium sensitizer, on myocardial calcium homeostasis during endotoxic shock in rats.
同时测定大鼠器官细菌易位率、器官易位肠杆菌计数、血浆内毒素等。
And the bacteria translocation rate and count in organs, and plasma endotoxin were determined.
目的研究大鼠休克复苏后小肠绒毛和血内毒素含量的改变及两者的相关性。
Objective to investigate experimentally the changes of intestinal mucosal morphology in correlation with endotoxin during shock and resuscitation after trauma.
目的探讨亚低温和高温对内毒素致急性肺损伤(ALI)大鼠肺微血管通透性的影响。
Objective to investigate the effects of mild hypothermia and hyperthermia on endotoxin-induced acute lung injury (ALI).
目的观察纳洛酮对内毒素刺激的大鼠离体支气管平滑肌细胞内皮素1和内皮素A受体基因表达的影响。
Objective To investigate the effect of naloxone on the expression of ET 1 and ET AR mRNA in cultured rat bronchial smooth muscle cells stimulated by LPS.
方法将12只雄性SD大鼠随机分为对照组和内毒素组。
Methods 12 male SD rats were randomly divided into control group and endotoxin group.
目的探讨分泌型白细胞蛋白酶抑制剂（SLPI）对内毒素（LPS）致新生大鼠急性肺损伤（ALI）的抗感染作用。
Objective To investigate the anti-inflammatory effects of secretory leukocyte protease inhibitor(SLPI) on newborn rats with lipopolysaccharide(LPS)-induced acute lung injury(ALI).
结论罗红霉素能改善大鼠急性肺损伤时气体交换能力，调节肺部细胞因子，对内毒素诱导的急性肺损伤有保护作用。
CONCLUSION Roxithromycin has a protective effect on rat acute lung injury induced by LPS through regulating cytokines, improving gas exchange ability, and inhibiting lung permeability.
虎杖甙对内毒素休克大鼠肺组织中磷脂酶a2的影响。
Effect of polydatin on phospholipase A2 in lung tissues in rats with endotoxic shock.
目的：观察内毒素刺激所致大鼠肝细胞溶酶体功能改变及下法对其影响。
Objective:To observe the effect of purgation methods on the changes of function of lysosome in rat liver cells in endotoxemia.
方法：观察双黄连注射液对内毒素休克小鼠的生存时间及对内毒素所致大鼠肝、肺、肾等病理改变的影响。
METHODS To observe the effect of Shuanghuanglian injection on the average death time of mice and the pathological changes of rats livers , lungs and kidneys induced by endotoxin.
目的比较异氟醚和异丙酚后处理对内毒素血症大鼠肺泡毛细血管屏障功能的影响。
Objective To compare the effects of posttreatment with isoflurane versus propofol on pulmonary alveolar capillary barrier (PACB) in endotoxemic rats.
目的：观察米诺环素对内毒素诱导的大鼠眼内炎症的抑制作用。
AIM: to investigate the inhibitory effect of minocycline on the ocular inflammation induced by lipopolysaccharide in the rats.
目的探讨内毒素（LPS）诱导的急性肺损伤（ALI）大鼠肺组织甲状腺转录因子-1（TTF-1）、肺表面活性物质相关蛋白-A（SP-A）表达的变化。
Objective To investigate the change of the expression of TTF-1 and SP-A in lung tissue of ALI rats induced by LPS.
前言：目的：研究内毒素作用于新生大鼠后，肺组织趋化因子-基质细胞衍生因子(CX CL12)表达的变化及可能作用。
Objective: To investigate the expression of CXCL 12 and its potential functions in neonatal rat lung caused by lipopolysaccharide (LPS).
目的：考察精制清开灵注射液对内毒素致大鼠多脏器损伤的保护作用。
Objective: to investigate the protecting effect of refined Qing Kai Ling (RQKL) on the damnify of rat viscera induced by endotoxin.
方法尾静脉注射内毒素制备ali大鼠模型。
Methods Rat ALI model was reproduced by injection of lipopolysaccharide into tail vein.
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