除了上述典型的过敏性坏死反应特征以外，寄主细胞还产生了细胞壁防御结构和次生物质。
Besides from the above-mentioned typical characters of hypersensitive reaction, the host cells also produced defense structure in cell walls and secondary metabolic materials.
它们被认为是生物反应的调节者，具有有激活巨噬细胞和T细胞的能力，并产生细胞因子、白细胞介素和肿瘤坏死因子。
They are reported to serve as biological response modifiers with the capability to activate macrophages and T-cells, and to produce cytokines, interleukins and tumor necrosis factors.
外科医生经常会在病人清醒的时候用电流刺激肿瘤周围的脑组织，观察病人的反应以避免切除那些没有坏死的部分。
Surgeons often electrically stimulate the area around the tumor while the patient is awake and can provide feeback, so they can avoid damaging critical tissue.
在该药上市后期间，已报道发生其他包括以硬结、蜂窝织炎、血肿、脓肿、无菌脓肿以及坏死为指征的注射部位反应事件。
In the postmarketing period, additional cases of injection site reaction with features including induration, cellulitis, hematoma, abscess, sterile abscess, and necrosis, have been reported.
镜下，移植物抗宿主反应是说明“程序性细胞死亡”或单个细胞坏死(即调亡)过程的最好例子。
Microscopically, graft versus host disease is one of the best examples of a process called "apoptosis" or single cell necrosis.
结果A 1组与B1组移植术后局部组织反应重，腱鞘形成生物屏障，影响肌腱的营养致使肌腱变性，纤维化，部分坏死。
Results in group A1 and B1, the local reaction was sever, the nutrition of tendon graft was barricaded by the whole sheath resulting in adhesion, degeneration and necrosis.
导致淋巴细胞和单个核细胞局部漫润等炎症反应甚至移植答官的坏死。
Causes the lymphocyte and the single nuclear cell inundates inflammations and so on Run to respond that partially even transplants answers official's necrosis.
上市后监测报告发现了重度肝脏不良反应病例，包括肝脏坏死、黄疸、暴发性肝炎伴或不伴黄疸、肝衰竭等。
Postmarketing surveillance reports include cases of severe hepatic reactions, including liver necrosis, jaundice, fulminant hepatitis with and without jaundice, and liver failure.
结果缺血性肠炎的病理改变以肠壁充血、水肿、出血及变性坏死为主，伴不同程度的炎症反应。
Results The main pathological features of IC are hyperemia, edema, hemorrhage, degeneration and necrosis of bowel wall with various degrees of inflammatory reaction.
在炎症细胞侵润的区域特别是坏死斑块的边缘，CRP反应较强。
At the region infiltrated by inflammatory cells, especially at the margin of necrotic plaque, CRP reaction is strong.
小鼠肌内埋置后植入部位无肌肉坏死，炎症反应轻，无纤维包裹。
The intramuscular implantation in mice induced no muscular necrosis or fibrous capsule formation, except for slight inflammatory reaction.
目的探讨肿瘤坏死因子- a (TNF - a)、去甲肾上腺素(NE)、内皮素(et)、肾上腺髓质素(adm)在全身炎症反应综合征(SIRS)病理过程中的作用。
Objective to investigate the role of tumor necrosis factor-a (TNF-a), norepinephrine (ne), endothelin (ET), adrenomedullin (ADM) in the pathogenesis of systemic inflammatory response syndrome (SIRS).
DH株系中的杀卵特性是通过叶鞘上杀卵反应产生的坏死症状表现的。
Ovicidal trait in DH lines was phenotyped by necrotic symptoms on the leaf sheaths due to ovicidal response.
脊髓损伤后所形成的坏死空洞和反应性增生的胶质瘢痕是导致脊髓损伤再生修复受阻的重要因素。
The formation of necrosis cavum and glial scar after spinal cord injured was one of important factor that affected regeneration of nerve fiber and neuron.
结果兔耳缘静脉滴注AZL未见对血管有明显的变性和坏死等严重刺激反应；
Results Serious stimulation such as denaturation and necrosis was not seen in rabbits' ear-rim auricular vein after given AZL.
在上市后监督后已报告的严重肝脏不良反应事件包括肝坏死、黄疸、伴有或不伴有黄疸的暴发性肝炎、肝衰竭。
Postmarketing surveillance has reported cases of severe hepatic reactions, including liver necrosis, jaundice, fulminant hepatitis with and without jaundice, and liver failure.
结果：移植肾术后早期少尿或无尿患者原因，急性排斥反应55.9%，急性肾小管坏死38.2%，加速排斥反应5.9%。
Results The reasons that led to oliguria or anuria early after transplantation were acute rejective reaction (55.9%), acute renal tubular necrosis (38.2%) and accelerated rejective reaction (5.9%).
活检病理结果慢性排斥反应2例，胆汁淤积性肝硬化1例，肝脏局灶性坏死1例。
The results of biopsy showed 2 chronic rejections, 1 biliary stasis hepatic cirrhosis and 1 hepatic focal necrosis.
结果显示单纯变态反应性疾病即可导致骨坏死发生。
The results revealed that osteonecrosis could be induced by simple allergic disease.
组织学观察发现淋巴细胞坏死，网状细胞增生等炎症反应。
Histopathological study showed necrosis and inflammation, such as depletion of lymphocytes, proliferation of reticular cells, etc.
结论术后影响移植肾成功的主要原因是急性排斥反应及急性肾小管坏死。
Conclusion The major causes of dysfunction of the transplanted kidney in the postoperative sfage are acute rejection and ATN.
全身炎症反应与一些细胞因子如内皮素、肿瘤坏死因子等的诱导表达有关。
SIR is relative to inducing and expression of some of cytokines such as ET and TNF .
冠心病组C-反应蛋白、肿瘤坏死因子、胰岛素、总胆固醇、低密度脂蛋白胆固醇、载脂蛋白B100均高于正常对照组，且有统计学意义（P<0.01，P<0.05）；
The serum levels of CRP, TNF, insulin, cholesterol(TC), low density lipoprotein cholesterol( LDLC) and apo B100 were much higher than those of the control group(P< 0.01, P<0.05).
埋植材料周围肌肉组织无脓肿形成和组织坏死发生，局部组织反应为非细菌性炎症反应。
The biodegradation pattern in vivo was similar to that in vitro. There was no abscess formation and tissue necrosis surrounding the implanted materials.
在进行手术前(除急诊外)，应尝试使用药物如柳氮磺吡啶、激素、硫唑嘌呤、肿瘤坏死因子拮抗剂及反应停。
Agents such as sulfasalazine, corticosteroids, azathioprine, TNFa antagonists and thalidomide should be tried first before surgery, except in emergencies.
本文提出，应该以促使肿瘤细胞坏死、诱导机体抗肿瘤免疫反应为抗肿瘤治疗的主导思想和研究方向。
The benefit of inducing apoptosis is not to cause inflammatory reaction, but as its disadvantage, it inhibits immune responses, and th…
本文提出，应该以促使肿瘤细胞坏死、诱导机体抗肿瘤免疫反应为抗肿瘤治疗的主导思想和研究方向。
The benefit of inducing apoptosis is not to cause inflammatory reaction, but as its disadvantage, it inhibits immune responses, and th…
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