近年来，肿瘤坏死因子受体超家族成员-OX40 (CD134)，作为一共刺激分子对于维持CD 4 +T细胞的活化、增值、存活起到关键性作用。
In recent years, tumor necrosis factor receptor (TNFR) family member-OX40 (CD134), as a key co-stimulatory molecule, involved in maintenance of activation, proliferation, survival of CD4 + t cells.
大多数雄激素非依赖性前列腺癌均高表达AR和核受体辅活化子、如转录中间因子2（TIF-2），ARA24、PIAS、类固醇受体辅活化子-1（SRC-1）等有力地支持了这个假说。
The "superactive AR"hypothesis was supported by the fact that the majority of AIPC expresses high level of AR and nuclear receptor(NR) coactivators, such as TIF-2, ARA24, PIAS, SRC-1.
应用四色荧光活化流式细胞术(FACS)检测CD 34 +细胞其粘附分子及趋化因子受体CXCR -4的表达谱。
Four color fluorescence activated cell sorting (FACS) analysis was applied to detect the expression profiles of adhesion molecules and chemokine receptor CXCR 4 on CD34 bright cells.
核受体是配体活化的转录因子，能调控大量的靶基因。
Nuclear receptors (NRs) are ligand-activated transcription factors that regulate the activation of a variety of important target genes.
核受体是配体活化的转录因子，能调控大量的靶基因。
Nuclear receptors (NRs) are ligand-activated transcription factors that regulate the activation of a variety of important target genes.
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