痛风是由于嘌呤代谢异常引起的疾病。
尿酸为嘌呤代谢产物，从尿中自由排出。
Uric acid is a product of purine metabolism that can excrete freely in urine.
痛风是长期嘌呤代谢障碍、血尿酸增高引起的代谢性疾病，高尿酸血症是痛风的重要生化基础。
Gout is a metabolic disease of tissue damage due to long-term purine metabolic disturbance, rising serum uric acid.
葡萄糖通过胰岛素依赖机制进入红细胞，大多数经代谢产生AT P，和还原型烟酰胺腺嘌呤二核苷酸(NADH)。
Glucose enters the RBC by an insulin-independent mechanism, and most is metabolized to produce ATP and reduced nicotinamide adenine dinucleotide (NADH).
尿酸是人体嘌呤碱基分解代谢终产物，经肾脏排泄。
Uric acid as the end-product of purine metabolism in human body is excreted through kidney.
结论：吗啡影响神经细胞嘌呤核苷酸代谢，使细胞内腺苷酸及腺苷水平增高，这可能是吗啡依赖和耐受形成的机理之一。
Conclusion morphine may affect purine nucleotide metabolism of nerve cells, increase the adenylate and adenosine levels, which may be one of the mechanism of morphine dependence and tolerance.
目的探讨丹曲林对缺血再灌注心肌嘌呤核苷酸代谢的影响。
Objective To investigate the effect of Dantrolene on purine nucleotide metabolism in myocardial ischemia-reperfusion.
观察大强度运动对大鼠嘌呤核苷酸代谢的影响，同时分析所建运动模型的可行性。
The effects of high intensity exercise on purine nucleotide metabolism of rats were studied in this paper. Simultaneously, the feasibility of the motor model was discussed.
观察大强度运动对大鼠血浆嘌呤核苷酸代谢的影响。
The effects of high intensity exercise on purine nucleotide metabolism of rats were studied in this paper.
还原型烟酰胺腺嘌呤二核苷酸(NADH)广泛存在于一切活细胞中，是细胞能量代谢所必需的辅酶。
Nicotinamide adenine dinucleotide reduced (NADH) is present in every living cell and is essential to cellular energy production.
尿酸是嘌呤的最终代谢产物。
Uric acid (UA) is an end product from purine derivatives in human metabolism.
本文探讨了小麦叶片生育后期衰老过程中的代谢变化以及抽穗期喷施氮肥、6—苄基腺嘌呤（6—BA）的影响。
This paper illustrates the metabolic changes of wheat leaves during laten period senescence, and the effects of spraying solution(2% urea and 10ppm 6-BA)at the time cf heading.
本文探讨了小麦叶片生育后期衰老过程中的代谢变化以及抽穗期喷施氮肥、6—苄基腺嘌呤（6—BA）的影响。
This paper illustrates the metabolic changes of wheat leaves during laten period senescence, and the effects of spraying solution(2% urea and 10ppm 6-BA)at the time cf heading.
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