测定小鼠T淋巴细胞功能，IL-2和血清丙二醛（MDA）水平。
The spleen T lymphocyte proliferation, IL-2 production and serum MDA were measured.
本实验通过大鼠脊髓损伤模型，观察了抗氧化剂对组织和血清丙二醛（MDA）的影响。
The effect of antioxidants on the malondialdehyde(MDA)in serum and spinal cord of rats was observed with rats'model of spinal cord injury.
测定血清中丙二醛（MDA）和超氧化物歧化酶（SOD）、乳酸脱氢酶（LDH）、磷酸肌酸激酶（CK）的含量。
Measured the contents of malondialdehyde(MDA), lactate dehydrogenase(LDH), creatine kinase(CK) and the activity of superoxide dismutase(SOD) in serum.
血清醌还原酶（QR）、ALT、LDH和CK催化活性用酶动力学方法测定，肝丙二醛（MDA）含量按硫代巴比妥酸方法测定。
Serum quinone reductase (QR), ALT, LDH and CK activities were determined with enzyme dynamic methods. Liver malondialdehyde (MDA) content was measured by the method of thiobarbituric acid.
方法：检测大鼠非致炎侧足爪体积，血清丙二醛(MDA)，滑膜细胞产生白细胞介素1 (IL 1)和亚硝酸盐(NO-2)量。
Methods: The volume of non-injected hind paw of AA rats, serum malondialdehyde (MDA) content, interleukin-1 (IL-1) and nitrite (no - 2) produced from articular synoviocytes were measured.
目的研究高压氧治疗脑梗死对血清超氧化物歧化酶和丙二醛活性的影响及其临床价值。
Objective To study the effect of hyperbaric oxygenation (HBO) therapy on the activities of serum SOD and MDA of patients with cerebral infarction and its clinical value.
目的探讨心肌缺血的家兔在急性低温低氧复合环境下血清超氧化物歧化酶（SOD）活力和丙二醛（MDA）含量的变化。
Objective To study the effects of acute low temperature and hypoxia on serum SOD and MDA of myocardial ischemia rabbit.
并检测血糖、糖基化血清蛋白和血清脂过氧化产物丙二醛浓度以反映代谢控制。
Serum concentrations of glucose, glycosylated serum protein, and malondialdehyde, derived from lipid peroxidation were also examined to estimate metabolic control.
方法用丙二醛(MDA)和一氧化氮(NO)试剂盒测定不同组别大鼠中度脑损伤后脑组织及血清中MDA和NO的含量。
Methods the content of malondialdehyde (MDA) and NO in the brain tissue and serum of rats in different group was examined.
主要观察指标：正常和心肌缺血家兔在急性低氧条件下血清sod和丙二醛水平比较。
MAIN OUTCOME MEASURES: Comparison of serum SOD and malondialdehyde between normal rabbits and myocardial ischemic rabbits under the acute hypothermia and hypoxia conditions.
测定血清丙二醛含量和总SOD活性以及肝组织匀浆中的脂褐素含量。
The content of MDA in serum and lipofuscin in liver homogenate and the activity of T SOD in serum were detected.
与模型对照组比较，山药合剂两剂量组血清谷丙转氨酶、谷草转氨酶和肝组织乳酸脱氢酶活性及血清葡萄糖、三酰甘油、游离脂肪酸和肝组织丙二醛含量显著降低（P<0.05～0.01）；
Compared with model control group, ALT, AST, Glu, TG, FFA in serum, and LDH activity and MDA content in liver tissues were markedly reduced in two Shanyao mixture groups (P < 0.05-0.01).
化学比色法分析血清丙二醛水平；采用MS302多媒体生物信号系统分别记录用药4和8周末大鼠坐骨神经传导速度。
MDA content in blood serum was analyzed by chromatometry and the speed of conduction in nerve was recorded in 4th and 8th week using the MS302 style of media bio-signal system.
检测各组患者血清丙二醛（MDA）水平和血清超氧化物歧化酶（SOD）活性，进行统计学分析和比较。
Serum malonaldehyde (MDA) level and superoxide dismutase (SOD) activity were detected in each group, and statistical analysis was conducted.
我们检测了小鼠血清活性氧(ROS)、总超氧化物歧化酶(T - SOD)、丙二醛(MDA)、谷胱甘肽过氧化物酶(GSH - PX)和E2水平。
The levels of serum reactive oxygen species (ROS), total superoxide dismutase (T-SOD), malondialdehyde (MDA), glutathione peroxidase (GSH-PX) and E2 were also detected.
紫外可见分光光度计测定血清游离脂肪酸（FFA）、丙二醛（MDA）和超氧化物歧化酶（SOD）的水平。
The levels of free fat acid (FFA), malonaldehyde (MDA) and superoxide dismutase (SOD) in serum were assayed by uv-vis spectrophotometer.
目的：测定妊娠期糖尿病(GDM)孕妇血清丙二醛(MDA)、超氧化物歧化酶(SOD)和一氧化氮(NO)含量，寻找监测gdm病情变化的临床指标。
Objective: the serum levels of malondialdehyde (MDA), superoxide dismutase (SOD) and nitric oxide (NO) were measured to find out the clinical index in GDM.
动态测定了常压间断低氧大鼠血清和肺组织丙二醛含量及红细胞和肺组织超氧化物歧化酶活性。
Serum and lung tissue Malondialdehyde (MDA) contents as well as erythrocyte and lung tissue Superoxide Dismutase (SOD) activities were measured during intermittent exposure to hypoxia in rats.
猪油组心线粒体膜、红细胞膜的膜流动性显著低于豆油组，血清及肝丙二醛水平亦显著低于麻油组和豆油组。
The heart mito- chodria membrane fluidity and red cell membrane fluidity of lard group rat were lower than th…
结果：JEXC能提高超氧化物歧化酶（SOD）的活性，降低心肌丙二醛（MDA）和血清肌酸酶（CK）含量。
Results: Jiu-Er-Xin capsules could increase the activities of SOD and decrease MDA content in myocardium.
结果：JEXC能提高超氧化物歧化酶（SOD）的活性，降低心肌丙二醛（MDA）和血清肌酸酶（CK）含量。
Results: Jiu-Er-Xin capsules could increase the activities of SOD and decrease MDA content in myocardium.
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