偶见吞噬体，胞浆内细胞器变性及大的泡状结构，内含絮状物。
The phagosome, degenerated organelles and big vacuoles-shape with flocculent material were also observed in the cytoplasm of bone marrow cells.
在尘细胞和吞噬碳粒细胞内，除含有吞噬体外还含有较多的自噬前体、自噬体和自噬溶酶体。
There were autophagosome precursors, autophagosomes, autophagolysosomes as well as phagosomes in the dust cells and the cells phagocytized carbon particles.
将淋巴结和吞噬碳粒后的细胞做超薄切片，观察巨噬细胞内的吞噬体、自噬体和溶酶体的结构和分布。
The ultrathin sections of the lymph nodes and the cells phagocytized carbon particles were prepared. The structures and distribution of phagosomes, autophagosomes and lysosomes were viewed.
到最后，吞噬细胞前体可能发生恶变，导致急性或慢性骨髓性白血病。
Finally, phagocyte precursors may undergo malignant transformation, which results in acute or chronic myelogenous leukemia.
然后迅速转化为血红蛋白二聚体与肝球蛋白质结合，并运送到肝脏经吞噬作用除去。
This is quickly converted to Hgb dimers that bind to haptoglobin and are transported to the liver, where they are metabolized in the same manner as products from RBC removed by phagocytosis.
巨噬细胞对钩体具有吞噬和杀伤作用，同时，致病性钩体可逃避巨噬细胞杀伤。
Macrophages can phagocytize and kill leptospira, while the pathogenic leptospira can evade the killing by macrophages.
目的研究豚鼠腹腔巨噬细胞吞噬钩端螺旋体毒力株的机制，探讨天然免疫在钩端螺旋体病发病机制中的作用。
Objective to investigate the mechanisms of phagocytosis of virulent Leptospira by peritoneal macrophages of guinea pigs, and evaluate the role of innate immune in the pathogenesis of leptospirosis.
目的研究豚鼠腹腔巨噬细胞吞噬钩端螺旋体毒力株的机制，探讨天然免疫在钩端螺旋体病发病机制中的作用。
Objective to investigate the mechanisms of phagocytosis of virulent Leptospira by peritoneal macrophages of guinea pigs, and evaluate the role of innate immune in the pathogenesis of leptospirosis.
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