目的：探讨创伤后多器官功能衰竭(MOF)患者血浆粘附分子和补体活化成分水平的变化及意义。
Objective: To evaluate the clinical significance of changes in cell adhesion molecules and composition of complement activation in trauma patients with multiple organ failure (MOF).
目的分析重型颅脑损伤后出现多器官功能衰竭(MSOF)的发生机理，探讨合理有效的治疗方法。
ObjectiveTo analyze the development mechanism and investigate the more effective therapeutic method of the multiple system organs failure (MSOF) after severe craniocerebral injury.
全身炎症反应综合症是创伤后机体常见的病理状态，是并发多器官功能衰竭的前提和基础。
Systemic inflammation response syndrome was one of frequent pathological conditions for post-trauma patients, which was the precondition and basis for multi-organs dysfunction.
凝血反应和炎症瀑布反应在心肺体外循环后可引起多器官功能衰竭。
Induction of the coagulation and inflammatory cascades can causemultiorgan dysfunction after cardiopulmonary bypass.
凝血反应和炎症瀑布反应在心肺体外循环后可引起多器官功能衰竭。
Induction of the coagulation and inflammatory cascades can causemultiorgan dysfunction after cardiopulmonary bypass.
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