最糟糕的病例包括大面积的肺损伤，可能是由于这个最强健群体存在过度的免疫反应所导致的。
The worst cases involve extensive lung damage, possibly as the result of an excessive immune response among the most robust segment of the population.
结论：脑出血早期存在多个差异表达基因，这些差异表达的基因可能在出血性脑损伤中发挥重要作用。
CONCLUSION: Early cerebral hemorrhage has many differential expression genes, which can play an important role in hemorrhagic brain damage.
结论SARS患者存在运动心肺功能障碍，可能与其心肺、肌肉等的损伤、发病后活动减少有关。
The patients of SARS have the dysfunction of cardiopulmonary exercise testing, which may be related with the injury of cardiopulmonary system from SARS-CoV and a few exercise.
在伤后12小时以内，损伤轴突尚未断裂，可能仍存在可逆性，这可能是临床上有效治疗的最佳时机。
Within 12 hours after injury, the injured axons were not yet devided, and its reversibility might still remain. This might be the most ideal time for effective treatment.
HCY在生理浓度铜离子存在下可能通过氧化应激损伤的机制而导致血管内皮细胞凋亡。
The apoptosis in vascular endothelial cells induced by HCY plus copper was involved in the mechanism of oxidative stress-mediated injury.
细胞免疫性肝损伤可能为HEV的主要致肝损伤机制，但不能排除存在病毒直接致肝损伤作用。
The immune mediated liver injury by lymphocytes might be a main pathogenesis of HE, but the liver injury induced directly by HEV might not be excluded.
由于它的存在可能与细胞损伤并存，TIGAR为科学家提供了一个重要的线索，指示癌症可能来临。
Because its presence can come in tandem with cellular damage, TIGAR is an important clue for scientists that could indicate cancer may follow.
结论：大鼠心肌组织存在钙敏感受体，其表达与月龄有一定关系，并可能参与心肌缺氧-再灌注损伤的发生。
CONCLUSION: Our results demonstrate that the CaSR is expressed in the rat cardiac tissue. The mRNA expression of CaSR changes with age and is involved in the anoxia-reperfusion injury.
地震前后的重力变化和波速变化以及电阻率变化可能也指示了深部岩体的结构损伤的存在。
The gravity, wave velocity and electrical resistivity changes before and after the earthquake may also be indicative of the deep rock of the existence of structural damage.
表明该场所的上述多综同时存在的危害物可对职业接触者的免疫功能有抑制作用，并可能使染色体细胞遗传物质损伤修复能力降低。
Indicate the place of the simultaneous presence of hazards in the occupation exposure of immune function inhibition, and may cause cell damage of genetic materials in lower repair capability.
表明该场所的上述多综同时存在的危害物可对职业接触者的免疫功能有抑制作用，并可能使染色体细胞遗传物质损伤修复能力降低。
Indicate the place of the simultaneous presence of hazards in the occupation exposure of immune function inhibition, and may cause cell damage of genetic materials in lower repair capability.
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