《公共医学图书馆摘要》：钙离子在活性依赖型的可塑性突触的讯息传递中具有关键性的地位。
PubMed abstract: Calcium ions play crucial signaling roles in many forms of activity-dependent synaptic plasticity.
Morikawa先生的研究(研究结果发表在上个月《神经科学杂志》上)表明，重复地暴露在酒精中，可以增加大脑中的一个关键部位突触的可塑性。
Mr Morikawa's study, results of which were published last month in the Journal of Neuroscience, found that repeated exposure to ethanol enhances synaptic plasticity in a key area in the brain.
研究团队观察到，发生SHANK3突变的神经细胞之间的沟通受损，他们还发现脑细胞和突触（神经细胞之间的接触点）的功能和结构的可塑性发生了变化。
The research team observed impaired communication between nerve cells in the mice with the SHANK3 mutation.
短时程突触可塑性包括短时程增强和短时程压抑两种类型。
Short - term synaptic plasticity includes two forms, short - term enhancement and short - term depression.
研究人员指出，酒精并不是通过杀死神经元细胞导致失忆发生的。实际上，是这些产生的甾体物质影响了突触可塑性，造成了LTP和记忆形成方面的损害。
The scientists point out that alcohol isn't causing blackouts by killing neurons. Instead, the steroids interfere with synaptic plasticity to impair LTP and memory formation.
因此通过理解突触的原始发育情形，我们也可能得以了解作为可塑性基础的分子机制。
So by understanding the initial development of synapses, we may also be getting at the molecular mechanisms underlying plasticity.
海马神经元损伤是神经突触可塑性调节中重要的一个环节，在抑郁症发病机制的研究中占有重要地位。
The damage of hippocampus neuron is the key part in plasticity regulation of synapse and play a critical role in the mechanism of depression.
一氧化氮作为一种气体信使分子，参与神经系统的信息传递，对海马、小脑等神经元突触的可塑性和神经网络的构建产生重要影响。
As a gas message molecule, nitric oxide takes part in the information transfer in nervous system, which affects plasticity of synapse in hippocampus, and cerebellum and construction of neural network.
突触可塑性对于脑发育过程中的神经环路重构以及学习记忆等脑的高级功能是非常重要的。
Synaptic plasticity is important for the remodeling of neural circuits during brain development and is implicated in higher brain functions such as learning and memory.
突触可塑性是神经系统所具有的重要特征，也是神经系统实现其功能的重要保障。
Synaptic plasticity is an important characteristic of neural system. It is significant to the function of neural system.
目的探讨短暂脑缺血再灌流后突触传递功能的改变和神经系统的可塑性。
Objective To investigate the synaptic transmission function and neural plasticity after transient ischemia followed by repufusion.
通过对出生前有慢性应激经历子代海马CA 1区LTP和LTD诱导性及学习记忆和行为反应的测定，探讨出生前慢性应激对子代海马CA 1区突触可塑性及学习记忆行为的影响。
We explored the effects of prenatal stress on hippocampal synaptic plasticity and behavior of the offspring by testing LTP and LTD in hippocampal CA1 area and learning and memory in Morris Water Maze.
大量实验数据证明，NR2B亚单位与哺乳动物的突触可塑性、学习记忆、痛觉和神经系统疾病有关。
Accumulating evidence indicates that, NR2B is crucial for synaptic plasticity, learning and memory and nervous system degenerative diseases.
记忆的保持——突触稳定性还是突触可塑性?。
Memory retention-the synaptic stability versus plasticity dilemma Wickliffe C.
已有广泛的研究表明突触可塑性如长时程增强和长时程抑制可以作为学习和记忆机制的细胞模型。
Extensive studies indicated that synaptic plasticity, such as long-term potentiation or long-term depression, is the cellular basis for learning and memory.
突触的数量和功能发生改变可引起突触可塑性的改变，进而影响学习记忆能力。
Changes in the number and function of synapsis can cause changes in synaptic plasticity, thereby can affect learning and memory.
近年来对NR2B研究表明，其在调控神经元突触的可塑性、学习与记忆以及治疗精神紊乱方面具有重要的意义。
The presence of NR2B plays the most important function among these subunits, NR2B has been utilized to modulate learning and memory, synaptic plasticity, and treating mental disorders.
突触可塑性常常影响突触结构的形状。
Synaptic plasticity often affects the shape of synaptic structures.
在中枢神经系统，CREB调节着神经细胞生长发育，参与神经细胞突触可塑性、长时程记忆的形成过程。
In central nerve system, CREB participates in promoting neuron growth, the processes of plasticity and long term memory.
有关的神经元突触在功能上具有一定的可塑性。
C and synaptic plasticity may exist in the respiratory neurons of B? T. C.
结论环境刺激对HIBD大鼠功能的促进作用可能与突触超微结构参数的改变，增强突触可塑性有关。
Conclusion The role of environmental stimulation in functional outcome of HIBD is likely to be related to change of synaptic substructure and increased synaptic plasticity.
目的观察大鼠脑梗死及电针干预后突触素(syp)的动态变化，探讨突触可塑性的物质基础、机制及针刺的影响。
Objective To explore the mechanism, material fundamental of synapse plasticity and acupuncture's efficacy by watching the alteration of SYP after cerebral infarction and acupuncture treatment.
腹侧被盖区多巴胺神经元的突触可塑性的改变可能在许多药物滥用(包括可卡因滥用)所导致的回路修饰中起着一定作用。
Changes in synaptic plasticity in dopamine neurons of the ventral tegmental area (VTA) may contribute to circuit modification induced by many drugs of abuse, including cocaine.
并且在不同发育期其对突触可塑性的损害不同，其中哺乳期是大鼠对PBDE209最敏感的时期。
We studied the effects of PBDE209 on synaptic plasticity in the DG in vivo, and discussed different effects of PBDE209 in the different developmental periods.
结果提示：一次性电击可以引起大鼠脑内突触界面结构发生可塑性变化。
The results suggest that one trial electric shock can cause synaptic interface structure in the brain of rats to come into being plasticity changes.
突触前可塑性是指通过对神经递质释放过程的干预、修饰，调节突触强度的过程。
Presynaptic plasticity is implicated in the modulation of the neurotransmitter release machinery and consequently in synaptic strength.
对影响认知的中枢胆碱能神经元的神经营养支持作用、抑制细胞凋亡、调节突触可塑性等可能是它们发挥保护作用的主要机制。
Neurotrophic factors can sustain the function of central cholinergic neurons as well as inhibit the neuronal apoptosis. They can also modulate the synaptic plasticity.
在本论文研究中，我们初步检测了酒精对于小脑突触可塑性的影响，实验结果表明酒精可以影响PF-PC突触LTD的表达。
In this dissertation, our preliminary data showed that alcohol could affect expression of PF-PC synaptic LTD when measuring the influence of alcohol on cerebellar plasticities.
已有的电生理学实验已经表明，褪黑素可以调节海马神经元的兴奋性和突触可塑性。
Previous electrophysiological experiments have revealed that melatonin modulates hippocampal neuronal excitability and plasticity.
而且，我们发现突触前灭活早老蛋白而突触后未灭活时改变了突触的短期可塑性和突触易化。
Moreover, we found that presynaptic but not postsynaptic inactivation of presenilins alters short-term plasticity and synaptic facilitation.
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