为了阻止血管紧缩素2受体而采用复合物治疗之后，研究人员发现通过阻止其中的受体之一at1rs——ACE2活动增加。
Following treatment with compounds to block both Angiotensin II receptors, the researchers found that by blocking one of these receptors - AT1Rs - ACE2 activity increased.
当该蛋白质复合物正常发挥作用时，它使这些受体退回到神经细胞内-在此这些受体无法接受或响应有利突触生长的信号-在合适的时间内。
When the protein complex is working properly, it moves the receptors back inside the nerve cell - where they can no longer receive and respond to the pro-growth signal - at the appropriate time.
脂多糖还能绑定到血浆白蛋白形成复合物，增加LPS的毒性以及LPS对膜受体的亲和力。
LPS can bind to plasma proteins and form complexes that increase toxic activity of LPS and affinity of LPS to cell receptors.
其原因在于受体介导的内摄作用能够增强细胞对合成复合物的摄取。
This was caused by an enhanced cellular uptake of the resultant complexes via a receptor-mediated endocytosis process.
Garcia说，这些结构，与我们在其他实验中做的同样好，表现出T细胞受体对异体和自体MHC -蛋白复合物使用完全不同识别机制。
"These structures, as well as other experiments we did, showed that the T-cell receptor USES a completely different recognition mechanism for the foreign and self MHC-protein complex," said Garcia.
合成多聚免疫球蛋白受体，受体-抗体复合物，生产及应用。
Synthetic poly-ig receptor, receptor-antibody complexes, production and use thereof.
尤其是，新的发现提示该蛋白质复合物使响应来自倍受研究的骨形态发生蛋白信号途径(BMP signaling pathway)的有利于突触生长的信号的受体解除任务。
In particular, the new findings suggest that the protein complex decommissions receptors that respond to pro-growth signals coming from the well-studied BMP signaling pathway.
表皮生长因子受体的三维结构通过同源蛋白模建的方法得到，而抑制剂和靶酶结合复合物结构则通过分子力学和分子动力学结合的方法计算得到。
The 3d structure of EGFR was constructed using homology modeling, and the complex structures between receptor and ligands were predicted by using molecular mechanics and molecular dynamics.
表皮生长因子受体的三维结构通过同源蛋白模建的方法得到，而抑制剂和靶酶结合复合物结构则通过分子力学和分子动力学结合的方法计算得到。
The 3d structure of EGFR was constructed using homology modeling, and the complex structures between receptor and ligands were predicted by using molecular mechanics and molecular dynamics.
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