目的研究参杞合剂在体外对CNE细胞周期及凋亡的影响。
Objective To investigate the inhibitory effect of SQ on CNE cells in vitro.
结果：模型组海马CA 1区细胞数明显减少，应用参知健脑方能减轻海马CA1区的损伤。
Result: the model rats number of cells in the CA1 district in hippocampus decreased. SHENZHIJIANNAOFANG could decrease the injure to the the CA1 district in hippocampus.
目的：评价参芪扶正注射液对乳腺癌术后患者化疗后细胞免疫状态影响及毒副反应。
Objective: To value the cellular immunity state of body and toxic effect of the treatment by chemotherapy combined with Shenqi-fuzheng injection used for breast cancer after operation.
生晒参—活血，加强细胞的吸收能力，促进生发。
Panax Ginseng - Helps to improve blood circulation, strengthen the cells absorptive capacity to promote hair growth.
目的观察参一胶囊对非小细胞肺癌的辅助治疗作用。
Objective: To study the effect of Shenyi capsule on patients with non-small cell lung cancer (NSCLC).
结论峨参石油醚提取物对肺癌细胞增殖具有明显的抑制及诱导凋亡的作用。
Conclusion the petroleum ether extracts of Anthriscus have obvious inhibitory effect on the proliferation of lung cancer cell, which can induce the cells' apoptosis.
目的探讨体外循环(CPB)心内直视手术对机体细胞免疫功能的影响以及参麦注射液的免疫调节作用。
OBJECTIVE To study the influence of Sen - Mei injection on cellular immune function of patients with CPB.
本文报道了云产三七根粗提物—生三七人参皂甙对小鼠骨髓细胞体外培养有促进细胞团增殖的作用。
This paper reports that raw ginsenoside, extract from panax notoginseng from Yunnan province, could promote the proliferation of routine bone marrow cells in vitro.
结果：参芪扶正注射液能（1）升高外周血细胞计数：治疗组小鼠外周血的红细胞、白细胞和血小板计数均高于对照组（P<0.05）。
Results: Shenqi Fuzheng Injection could (1)increase peripheral blood cell count: the count of RBC, WBC and PLT in therapy mouses are higher than in the control ones (P<0.05).
目的观察比较参芪抑癌液、细胞因子对晚期肝、胰腺癌治疗效果。
Objective To compare the effect of Shenqi Suppressor Carcinoma Liquid and Cytokine on late liver cancer and pancreas cancer.
结论：参麦注射液可能通过调节pdgf表达对脑出血后迟发性神经细胞损伤修复有保护作用。
Conclusion: SM injection might attenuate the delayed injury induced by intracerebral hemorrhage via regulating the expression of PDGF.
五加双参片组骨髓微核细胞千分率低于模型对照组（P＜0.01）；
The percentage of the micronuleated cells in the WJSST groups was markedly lower than that in model group (P<0.01).
目的：观察参附汤对移植后小鼠造血干细胞归巢的影响。
Objective: to observe the effect of Ginseng and Aconiti Decoction to homing of hemopoietic stem cells of transplanted mice.
近几年来，国际上对于细胞运动分子机制的研究取得很大进展，利用组织培养细胞进行的大量的体外实验证明，皮层蛋白很可能直接参于细胞运动的产生过程。
Recently, great progress has been made in understanding of the mechanism of cell movement in vitro, and probably, cortactin acts directly on the molecular machines of cell motility.
目的观察参附汤对移植小鼠造血干细胞表面CD44及L-选择素（CD62L）的影响。
Objective To observe the effect of Ginseng and Aconiti Decoction on the expressions of CD44 and CD62L on surface of hematopoietic stem cells in transplanted mice.
细胞色素P 450和谷胱甘肽s转移酶是二组参于抗肿瘤药物和致癌剂代谢的重要酶系。
Cytochrome P450 and GST are two important complex gene families of enzymes that metabolize therapeutic drugs and carcinogens.
研究表明，经过炮制的红参，会产生一般生晒参不具有的五种人参皂甙特殊成份，而这些成份则具有抑制癌细胞的作用。
The results show that, after processing of red ginseng, ginseng produces regular students do not have the special ingredient of five ginsenosides, and these components are inhibited cancer cells.
目的：观察参脉注射液与刺五加注射液治疗老年慢性支气管炎患者T细胞亚群的变化。
Object: To observe the changes of t cell subpopulation of chronical bronchitis in senile people treated by ginseng ophiopogon injection and acanthopanax injection.
结论参莲方提取物可明显改善模型大鼠血液流变学指标，在改善血管内皮细胞功能方面优于参莲方汤剂。
Conclusion Shenlianfang extracts can significantly improve the hemorheology parameters of model group rats, and has better effects on improving vascular endothelial cell function than the decoction.
目的观察参附注射液对健择联合顺铂方案（GP）治疗非小细胞肺癌的减毒作用。
Objective To observe the detoxification of Shenfu Injection(SHF) on Gemcitabine plus Cisplatin(GP) for treating non-small cell lung cancer(NSCLC).
目的：探讨三参滋胃饮对慢性萎缩性胃炎（CAG）大鼠胃黏膜G细胞和D细胞的作用及其机制。
Objective To study the effects and mechanisms of Sanshen Ziwei Yin(SZY) on D cells and G cells of gastric mucosa in rats with chronic atrophic gastritis (CAG).
目的：观察参附汤对移植后小鼠造血干细胞归巢的影响。
To explore the effect of glucose fluctuation on vascular cells and kidney cells.
实验结果显示：MPTP慢性帕金森病模型中，细胞凋亡参于了PD的发病机制。
The test prompted that apoptosis attended in mechanisms of PD in MPTP chronic parkinsonism model.
实验结果显示：MPTP慢性帕金森病模型中，细胞凋亡参于了PD的发病机制。
The test prompted that apoptosis attended in mechanisms of PD in MPTP chronic parkinsonism model.
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