结论：GLUT1在缺血半影区的表达上调，可能是机体对缺血损伤的保护性反应。
CONCLUSION: GLUT1 expression is notably up-regulated in the penumbra region after focal cerebral ischemia, it may be a protective reaction against ischemic injury.
而应用针刺治疗后各组缺血坏死灶范围减小，半影区炎性细胞粘附及浸润的程度明显减轻。
After treated by acupuncture, the necrosis areas reduced and the degree of the adhesiveness and infiltration of the inflammatory cells also decreased.
与H0缺血组小鼠皮层半影区相比，H4缺血组小鼠皮层半影区内PKCII和膜转位水平明显增高 （p<0.05）。
Whereas cPKC II and membrane translocation levels in penumbra of ischemic cortex in H4+Ischemia group increased significantly compared with that of mice in H0+Ischemia group (p<0.05).
与H0缺血组小鼠皮层半影区相比，H4缺血组小鼠皮层半影区内PKCII和膜转位水平明显增高 （p<0.05）。
Whereas cPKC II and membrane translocation levels in penumbra of ischemic cortex in H4+Ischemia group increased significantly compared with that of mice in H0+Ischemia group (p<0.05).
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