肾功能障碍、纤维肌痛和动脉粥样硬化(即由于胆固醇堆积至动脉缩小)，这些身理病变均可导致性冷淡。这项研究发现。
Kidney disorders, fibromyalgia and atherosclerosis (the narrowing of arteries due to cholesterol buildup) are all associated with anorgasmia, the review found.
巨噬细胞是动脉粥样硬化的发病机制的组成部分，在炎症和胆固醇平衡运作，其生物学特性的一个重要课题。
The macrophage is integral to the pathogenesis of atherosclerosis, functioning at the intersection of inflammation and cholesterol homeostasis, making its biological properties an important topic.
在第二个研究中，研究者测量hdl外排，这是一个病人的高密度脂蛋白(HDL)从参与动脉粥样硬化的细胞中清除胆固醇的能力。
In the second study, the researchers measured HDL efflux, which is the ability of a patient's HDL to remove cholesterol from cells involved in atherosclerosis.
图示：动脉粥样硬化高倍镜显示多量泡沫细胞，偶见胆固醇结晶(棱状空隙)。一些暗蓝色的炎细胞散在分布于粥样硬化病灶内。
This high magnification of the atheroma shows numerous foam cells and an occasional cholesterol cleft. A few dark blue inflammatory cells are scattered within the atheroma.
作者写道，在动脉粥样硬化斑块进展超过一个特殊的点以前，通过降低有害的低密度脂蛋白胆固醇水平能起到预防作用。
The authors write about prevention through reducing levels of bad LDL cholesterol before atherosclerotic plaque has progressed beyond a particular point.
方法：应用高胆固醇饮食建立兔动脉粥样硬化模型。
Methods: the atherosclerotic model was established in rabbits with high cholesterol diet.
虽然动脉粥样硬化比较常见，但是胆固醇栓子是少见的，至少是大多情况下不明显的。
Despite the frequency of aortic atherosclerosis, cholesterol emboli are rare, or at least insignficant most of the time.
高密度脂蛋白受体在胆固醇逆向运输中起重要作用，并且其可以做为动脉粥样硬化的新型治疗靶点。
HDL receptor plays a very important role in reverse cholesterol transport, and it represents a novel therapeutic target for atherosclerosis.
动脉粥样硬化的预言者ldl胆固醇脂：来自人类和动物的脂质饮食研究的证据。
LDL cholesteryl oleate as a predictor for atherosclerosis: evidence from human and animal studies on dietary fat.
对正常、高胆固醇血和动脉粥样硬化家兔主动脉结构糖蛋白（SGP）进行了比较。
Some properties of the aortic SGP from the normal, hyper-cholesterolemia and atherosclerotic rabbits were studied chemically andelectrophoretically.
方法：采用雄性日本鹌鹑喂饲高胆固醇膳食，造成动脉粥样硬化动物模型，动态观察主动脉壁的中内膜的多胺变化。
Method: the male Japanese quails which had high cholesterol diet were made into the models of atherosclerosis, and the polyamine changes of their aortic media-intima were dynamically observed.
目前，他正在研究糖尿病、肥胖症和高胆固醇是如何导致动脉粥样硬化疾病的。
He is currently studying how diabetes, obesity, and high cholesterol lead to atherosclerosis.
哥本哈根大学医院的研究人员称，这“难以解释”为众所周之的低密度脂蛋白或胆固醇可导致阻塞动脉的动脉粥样硬化。
The researchers at Copenhagen University Hospital said this was "difficult to explain" as LDL, or bad, cholesterol is known to cause atherosclerosis which can block arteries.
“这项发现允许我们将动脉粥样硬化和相关的状况如糖尿病和高胆固醇分开，”他说。
"This discovery allows us to separate atherosclerosis from associated conditions such as diabetes and high cholesterol," he says.
动脉粥样硬化斑块中的胆固醇主要来自LDL,LDL - C可以作为评价稳定型心绞痛患者预后的客观物质指标。
Atherosclerotic plaques of cholesterol mainly from LDL, LDL-C can be used as evaluation of the prognosis of patients with stable angina objective material indicators.
提示血清丙二醛、总胆固醇和甘油三酯的海拔性变化，可能是动脉粥样硬化海拔性差异形成的主要原因之一。
It indicated that the altitude changes of MDA, TC and TG could be an important factor of altitude difference in atherosclerosis.
动脉粥样硬化(as)是严重威胁人类的疾病之一，氧化型胆固醇被认为是AS的主要致病因素。
Atherosclerosis (as) is one of the most serious diseases in human, and the cholesterol oxidation products (Ch-Ox) are currently considered as a key atherogenic factor in its pathogenesis.
结果番茄红素可显著降低大鼠血清中胆固醇（TC）、甘油三酯（TG）、低密度脂蛋白胆固醇（LDLC）水平及动脉粥样硬化指数（AI）（P<0.05），适当升高高密度脂蛋白胆固醇（HDLC）水平；
The pathological changes in liver were observed by HE staining method. Results Serum and liver TC, TG, LDLC, AI levels in the three lycopene groups notably decreased (P<0.05), and HDLC increased.
克隆小猪模型去检验高脂肪、高胆固醇饮食诱导动脉粥样硬化。
A cloned pig model for examining atherosclerosis induced by high fat, high cholesterol diets. .
胆固醇逆转运（RCT）是机体排出过多胆固醇的唯一途径，也是HDL抗动脉粥样硬化的重要机制之一。
Recent studies revealed that the antiatherosclerotic effect of HDL was mainly attributed to its role in reverse cholesterol transport(RCT).
结论：茶多酚能增加实验性A S兔肝组织中hl脂酶活性，这一作用与降低血浆胆固醇水平和抗动脉粥样硬化密切相关。
CONCLUSION: TP increased HL activity in liver tissue in as rabbits, which correlated with decreasing the TC and LDL-c level in plasma and the area of atherosclerosis plaque.
结论：茶多酚能增加实验性A S兔肝组织中hl脂酶活性，这一作用与降低血浆胆固醇水平和抗动脉粥样硬化密切相关。
CONCLUSION: TP increased HL activity in liver tissue in as rabbits, which correlated with decreasing the TC and LDL-c level in plasma and the area of atherosclerosis plaque.
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