因此，我们现在将注意力放在当代的动脉粥样化形成理论上。
Thus we now turn our attention to contemporary theories of atherogenesis.
尽管由实验模型得到的数据支持CRP在动脉粥样硬化形成中的作用，但它的直接关系仍在讨论中。
Although some data from experimental models support a role of CRP in atherogenesis, its direct involvement remains in question.
冠状动脉钙化(CAC)是冠状动脉粥样硬化形成的早期特征，是冠心病(CHD)的重要特点。
Coronary artery calcification (CAC) is an important characteristic of coronary heart disease (CHD), which is also an earlier sign of atherosclerosis.
图示：冠状动脉粥样硬化形成血栓，是动脉粥样硬化的另一个复合病变。可见冠状动脉前降支有暗红色的血栓形成。
This is coronary thrombosis, one of the complications of atherosclerosis. The dark red thrombus is seen in the anterior descending coronary artery.
结论EGB对于动脉粥样硬化形成及经皮腔内冠状动脉介入治疗后再狭窄的防治可能具有应用前景。
CONCLUSION EGB may have some potential clinical implication in the prevention and treatment of atherosclerosis and restenosis after percutaneous transluminal coronary angioplasty.
在动脉粥样硬化形成的炎症机制中，C反应蛋白(CRP)起着重要作用。
C-reactive protein (CRP) plays an important role in the inflammatory mechanisms of atherosclerotic formation.
艾滋病毒是通过相关的免疫激活，慢性炎症，凝血功能障碍，和血脂紊乱机制，促进动脉粥样硬化形成。
HIV is known to promote atherosclerosis through mechanisms related to immune activation, chronic inflammation, coagulation disorders, and lipid disturbances.
先天免疫反应的激活在动脉粥样硬化形成中具有重要作用。
It now appears that activation of innate immune responses play a major role in atherosclerotic lesion development.
动脉中管壁的脉动低切应力在动脉粥样硬化形成中起始动和主要的决定作用。
The pulsatile low wall shear stress of arteries plays a initiated and major role in the developing of Atherosclerosis.
现就MMP - 9在动脉粥样硬化形成中机制及临床意义作一综述。
This article provides a brief review of the mechanism and clinical significance of MMP-9 in atherosclerosis and acute coronary syndrome.
我们假设PARP可能在动脉粥样硬化形成中起重要作用，它的抑制可能会减弱动脉粥样硬化实验模型中动脉硬化斑块的进展。
We hypothesized that PARP may play an important role in atherogenesis and that its inhibition may attenuate atherosclerotic plaque development in an experimental model of atherosclerosis.
我们推测动脉粥样硬化形成过程中血管平滑肌细胞能量代谢发生了改变。
During atherogenesis, vascular smooth muscle cells proliferate, migrate from tunica intima to media and uptake lipid then become foam cells.
目的：探讨动脉粥样硬化形成与血管平滑肌细胞端粒酶活性表达的相关性。
AIM: To observe the relationship between aortic atherosclerosis plaque formation and telomerase activation in vascular smooth muscle cells (VSMC).
在动脉粥样硬化形成的初级阶段，血小板在一定程度上诱导了斑块的形成，而在晚期的并发症中也起了重要作用。
In the initial stage of atherosclerosis, platelets induce the formation of atherosclerosis plaques, while they also contribute to the complications of atherosclerosis in the later stage.
血小板在动脉粥样硬化形成过程中的重要作用也日益被人们所认知并接受。
Moreover, platelet and its significant role in the atherosclerosis pathogenesis have drawn more and more attention.
目的研究高脂间歇低氧诱发动脉粥样硬化形成的重要作用基因。
Objective To investigate gene expression patterns of atherosclerosis (As) induced by intermittent hypoxia and hyperlipemia.
目的：观察2型糖尿病患者餐后甘油三酯水平对动脉粥样硬化形成的影响。
Objective: To study the influence of post-prandial hypertriglyceridemia on atherosclerosis in type 2 diabetic patients.
图示：动脉分支的上方有个大动脉粥样硬化形成的动脉瘤。
Here is an example of an atherosclerotic aneurysm of the aorta in which a large "bulge" appears just above the aortic bifurcation.
图示：动脉分支的上方有个大动脉粥样硬化形成的动脉瘤。
Here is an example of an atherosclerotic aneurysm of the aorta in which a large "bulge" appears just above the aortic bifurcation.
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