治疗冠状动脉痉挛可能涉及钙拮抗剂。
The treatment of coronary artery spasms may involve a calcium antagonist.
背景：动脉痉挛是SAH后临床早期常见并发症。
BACKGROUND: Vasospasm is a frequent complication in the early clinical course after SAH.
动脉痉挛引起动力性冠状动脉狭窄并产生的疼痛，虽然是缺血性的，也往往在休息或夜间发生。
Pain due to dynamic coronary narrowing from arterial asm, although ischemic, tends to occur at rest or nocturnally .
冠状动脉内肌肉痉挛会导致心脏病发作。
A muscular spasm in the coronary artery can cause a heart attack.
研究者们将米力农注入受血管痉挛所影响的脑区的动脉中。
The investigators infused milrinone intra-arterially into the cerebral territories compromised by the vasospasm.
实验证明，当小动脉发生持续性收缩时，骨胳肌发生痉挛跳动，肢体麻木等现象。
Experiments have proved that when the arteriole produce durative contraction, skeletal muscle cramps occur, numbness, etc.
结论：脑梗与动脉瘤性SAH后的不良预后显著相关。症状性血管痉挛是最重要的与脑梗相关的有可能被治愈的因素。
CONCLUSION: Cerebral infarction was strongly associated with poor outcome after aneurysmal SAH. The most important potentially treatable factor associated with infarction was symptomatic vasospasm.
冠状动脉血管痉挛能造成一个暂时的，突然的，显着的心外冠状动脉内径的减少。
Coronary artery vasospasm can cause a transient, abrupt, marked decrease in the diameter of an epicardial coronary artery.
但大动脉CVS表现无法完全解释如“有血管痉挛无症状”和“有症状而无血管痉挛”等临床现象，最终的影响因素可能是微循环改变。
Not all clinical findings can be well explained by CVS of major arteries, such as ischaemia without CVS, or CVS without ischaemia, the real determinant factors may relate to cerebral microcirculation.
两名病人在第二次动脉内输注米力农后血管痉挛获得缓解，三名病人则常规接受了血管成型术。
Two were resolved successfully after a second intra-arterial infusion of milrinone and three underwent mechanical angioplasty.
因动脉瘤破裂造成的蛛网膜下腔出血主要引发血管痉挛，其次才是团块损害。
The subarachnoid hemorrhage from a ruptured aneurysm is more of an irritant producing vasospasm than a mass lesion.
NO具有扩张血管平滑肌、松弛支气管平滑肌及降低肺动脉压、消除支气管痉挛的作用。
NO can lower the pressure of pulmonary artery and relieve the bronchospasm through dilating the smooth muscle of blood vessel and relaxing the smooth muscle of bronchus.
目的为寻求一种防治破裂脑动脉瘤急诊术后脑血管痉挛的效果确切、操作简便的方法。
Objective To find an effective and simple method of preventing cerebral vasospasm after emergency operation of ruptured intracranial aneurisms.
目的总结动脉瘤手术后血管痉挛及延迟性缺血性神经功能障碍(DIND)的防治经验。
Objective to summarize the experience of treating postoperative cerebral vasospasm and related delayed ischemic neurological deficit (DIND) of intracranial aneurysm.
腿部动脉血管病变最常见的是跛行，一种由于供血不足影响到肌肉而导致的痉挛性疼痛。
The most common symptom of arterial disease of the leg is claudication, a cramping pain caused by an inadequate supply of blood to the affected muscle.
动脉瘤性蛛网膜下腔出血一项主要并发症是症状性血管痉挛，它是排除了其他原因的缺血之后的一组神经损伤复合症候群。
A major complication of aneurysmal subarachnoid hemorrhage (SAH) is symptomatic vasospasm, a complex syndrome consisting of neurological deterioration and exclusion of other sources of ischemia.
基底病理学检查进一步证实血管痉挛的发生通过动脉灌注治疗，可以显著减少CVS引起的脑神经细胞凋亡。
The pathological study revealed that the selective arterial perfusion of nimodipine could reduce the apoptosis of cerebral nerve induced by CVS.
基底病理学检查进一步证实血管痉挛的发生通过动脉灌注治疗，可以显著减少CVS引起的脑神经细胞凋亡。
The pathological study revealed that the selective arterial perfusion of nimodipine could reduce the apoptosis of cerebral nerve induced by CVS.
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