除了炎症反应外，血小板通过释放生长因子和趋化因子刺激修复得以募集循环中血管内皮组细胞。
In addition to inflammation, platelets stimulate repair by releasing growth factors and chemokines to recruit circulating vascular progenitor cells.
血流淤滞：由于血管扩张、炎症渗出，使血流变慢，这有利于炎症介质和炎细胞聚集引起炎症刺激。
Vascular stasis: slowing of the blood in the bloodstream with vasodilation and fluid exudation to allow chemical mediators and inflammatory cells to collect and respond to the stimulus.
PAF在许多生物学过程中，如前炎症细胞的信号传导和活化、改变血管通透性、刺激糖原代谢以及其它方面起着重要的作用。
PAF has been implicated as a mediator of physiological processes such as signaling and activation of proinflammatory cells, alteration of vascular permeability, and stimulation of glycogen metabolism.
炎症反应中的细胞类型随炎症灶的时相和刺激物的不同而有差异。
The cell type present in the inflammatory response varies with the age of the inflammatory lesion and with the type of stimulus.
免疫毒性的作用机制可能通过氧化、炎症刺激和神经性炎症反应等有关，但具体致病机制仍不清楚，有必要从整体上研究颗粒物的毒性及其作用机制。
Damage of fine particulate is through oxidant stress, inflammation and neurogenic inflammation and so on, but the detail of the mechanism for them is lacking.
治疗湿疹有三步，保持宝宝的肌肤充足的水分，治疗炎症，识别和远离任何潜在的刺激源。
Treating eczema is a three-part plan: Keep baby's skin well moisturized, treat the inflammation, and identify and remove any potential triggers.
神经生长因子水平炎症过程中的神经生长因子增加外源性和管理导致痛觉过敏，过敏热刺激和肌肉痛。
NGF levels are increased in inflammatory processes and administration of exogenous NGF leads to hyperalgesia, hypersensitivity to thermal stimulation and muscular pain.
神经生长因子水平炎症过程中的神经生长因子增加外源性和管理导致痛觉过敏，过敏热刺激和肌肉痛。
NGF levels are increased in inflammatory processes and administration of exogenous NGF leads to hyperalgesia, hypersensitivity to thermal stimulation and muscular pain.
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