结论：触发或自律性异常是再灌注心律失常的发生机制，钙离子超负荷发挥作用。
Conclusion: Trigger activity or increased automaticity is one of mechanisms of reperfusion arrhythmia, and calcium overload plays an important role.
急性心梗发病后，它可导致心梗范围扩大、再灌注时局部心肌微循环衰竭以及提高室性心律失常的发生率。
It may extend the infarct size, contribute to the no-reflow phenomenon after reperfusion, and cause a higher incidence of ventricular arrhythmias.
结果表明益气活血方能显著降低急性心肌缺血及再灌注时心律失常的发生率及严重性。
The results demonstrated that qi-tonics and blood-activating decoction could reduce significantly the incidence and severity of arrhythmias caused by myocardial infarction and ischemia-reperfusion.
方法以扎、松冠状动脉左前降支的方法制作25只犬缺血-再灌注室性心律失常模型，以针电极探查希氏-浦肯野系统（HPS）的电冲动。
Methods The electrical activity from the His-Purkinje system (HPS) was recorded by plunge electrodes along the anatomic course of HPS in 25 dogs.
方法以扎、松冠状动脉左前降支的方法制作25只犬缺血-再灌注室性心律失常模型，以针电极探查希氏-浦肯野系统（HPS）的电冲动。
Methods The electrical activity from the His-Purkinje system (HPS) was recorded by plunge electrodes along the anatomic course of HPS in 25 dogs.
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