术后角膜内皮细胞密度三组之间无差异。
After surgery the three groups of corneal endothelium density had no statistical difference.
分别观察两组术前及术后的角膜内皮细胞密度。
To observe the corneal endothelium cell density before and after cataract surgery.
主要指标角膜内皮细胞密度和内皮细胞层CMTF形态。
Main Outcome Measures endothelial density and endothelial morphological images with CMTF.
结果玻璃体视网膜手术可以引起角膜内皮细胞密度的下降。
ResultsThe corneal endothelial cell densities were decreased following all kinds of surgeries.
结论LASIK术后早期不引起中央部角膜内皮细胞密度和形态改变。
Conclusion LASIK doses not induce changes in the density and morphology of the corneal endothelial cells in the early stage after operation.
目的：探讨角膜内皮细胞密度在超声乳化白内障吸除术以及小梁切除术治疗原发性闭角型青光眼前后的变化。
AIM: to compare the changes of corneal endothelial cell density before and after phacoemulsification and trabeculectomy treating the patients with primary angle-closure glaucoma.
所有患者均应用非接触型角膜内皮显微镜观察和测量角膜内皮细胞密度、六角形细胞的百分数、平均细胞面积等各项指标。
Cell density, percentage of hexagonal cells, and average cellular area of the corneal endothelium were determined by a non-contact specular microscope and a computer-aid image analysis system.
方法利用低密度脂蛋白作用人血管内皮细胞系ecv304，通过基因芯片分析人信号传导相关蛋白的表达谱。
Methods Microarray analysis on human signal transduction associated proteins was applied to profile changes in gene expression of human ECV304 endothelial cells induced by low density lipoprotein.
发生过程中内皮细胞由厚变薄，血管平均截面积逐渐减小，管腔的体密度增大；
During development, the endothelial cells thinned , the average profile area of the vessels reduced and the volume density of vascular lumen increased gradually.
分别以微机图像分析系统和人工计数对微血管内皮细胞面积(MEA)和微血管密度(MVD)进行定量分析。
Microvessel endothelial area (mea) and microvessel density (MVD) were determined by means of computer aided image analysis system (CIAS) and manual count.
用CD 34标记瘤组织血管内皮细胞，测定微血管密度(MVD)。
The endothelial cells of blood vessels within the tumor were labeled by CD34 for calculating MVD.
本文报道应用非接触式角膜内皮显微镜，观察107例不同年龄正常人角膜中央部内皮细胞的形态，密度和面积的结果。
This paper reports the shape, density and size of endothelial cells of central cornea which were observed under noncontact specular microscope in 107 normal persons with a wide range age.
使用角膜内皮显微镜观察手术前后角膜内皮细胞的平均密度、六边形细胞所占百分率。
The average density of endothelial cells and the percentage of hexagonal cells of corneal epithelium were observed before and after operations.
目的探讨血凝素样氧化低密度脂蛋白受体1 (LOX - 1)在氧化低密度脂蛋白(ox - LDL)诱导血管内皮细胞粘附分子表达中的作用。
Objective to investigate the effects of oxidized low-density lipoprotein receptor 1 (LOX-1) on secretion of adhesive molecules mediated by ox-LDL in human umbilical endothelial cells (HUVECs).
结论LASIK手术对角膜内皮细胞无损伤，不会造成角膜中央部内皮细胞形态改变和密度降低。
Conclusion LASIK is scatheless to cornea endothelial cells, and can't bring modality change and density decrease.
目的研究氧化低密度脂蛋白对人脐静脉内皮细胞表达CD 40中血凝素样氧化型低密度脂蛋白受体1的作用。
Objective To investigate the action of LOX-1 in Oxidized Low-Density Lipoprotein induced the expression of CD40 in cultured human umbilical vein endothelial cells.
目的：观察4 ~ 30岁青少年儿童角膜内皮细胞的密度和形态特点及内皮细胞随年龄变化的规律。
AIM: to observe the cell density and morphometric characteristics of corneal endothelia in 4-30 years old children and youngsters and to estimate the changes in corneal endothelia during this period.
目的：拟验证脐静脉内皮细胞低密度种植于脱细胞猪动脉支架实现血管内皮化的可行性。
OBJECTIVE: To explore endothelialization of low density human umbilical vein endothelial cell (HUVEC) seeded on decellularized porcine carotid arteries scaffold.
正常青年和儿童的角膜内皮细胞呈六角形，大小一致；而40岁以上者形态和大小不一致。内皮细胞的密度随年龄增长而减低，面积随年龄增长而增加。
The normal corneal endothelial cells of youths and children were hexagonal in shape and uniform in size, but they became irregular both in shape and size in persons above 40 years old.
免疫组织化学染色观察创面组织中新生血管内皮细胞表面标志物CD31表达，计算微血管密度。
The expression of CD31, surface marker of neovascular endothelial cells, was detected within the wound sites by immunohistochemical staining, and the microvessel density was calculated.
凝集素样氧化低密度脂蛋白受体(LOX - 1)是内皮细胞膜上特异性识别摄取ox - LDL的最主要受体，LOX - 1在介导ox - LDL导致内皮功能紊乱的过程中起关键作用。
LOX-1 was identified from endothelial cells as the main receptor specially recognizing and uptaking the ox-LDL, LOX-1 plays a key role in the endothelial dysfunction triggered by ox-LDL.
凝集素样氧化低密度脂蛋白受体(LOX - 1)是内皮细胞膜上特异性识别摄取ox - LDL的最主要受体，LOX - 1在介导ox - LDL导致内皮功能紊乱的过程中起关键作用。
LOX-1 was identified from endothelial cells as the main receptor specially recognizing and uptaking the ox-LDL, LOX-1 plays a key role in the endothelial dysfunction triggered by ox-LDL.
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