目的：探讨骨髓间充质干细胞体外扩增及其向内皮样细胞定向分化条件。
AIM: to explore the conditions of isolation, expansion and differentiation of bone marrow mesenchymal stem cells (MSCs) into endothelium like cells in vitro.
目的研究氧化低密度脂蛋白对人脐静脉内皮细胞表达CD 40中血凝素样氧化型低密度脂蛋白受体1的作用。
Objective To investigate the action of LOX-1 in Oxidized Low-Density Lipoprotein induced the expression of CD40 in cultured human umbilical vein endothelial cells.
血管内衬胞质丰富、深嗜伊红染的上皮样内皮细胞。
The vessels were lined by epithelioid endothelial cells with abundant eosinophilic cytoplasm.
目的探讨复方莶草合剂抗动脉粥样硬化内皮细胞损伤的作用。
Objective To study the effects of this mixture on anti-endothelial cell injuries.
目的：探讨蜈蚣对动脉粥样硬化家兔血管内皮细胞功能的影响。
Objective: To explore the effect of Centipede on vascular endothelial cells function in atherosclerosis rabbit.
目的观察两种木贼提取物对动脉粥样硬化早期内皮细胞超微结构的影响。
Objective To investigate the effect of Equisetum extract on the ultrastructure of endothelial cell in hyperlipidemia rats on early atherosclerosis stage.
目的探讨血凝素样氧化低密度脂蛋白受体1 (LOX - 1)在氧化低密度脂蛋白(ox - LDL)诱导血管内皮细胞粘附分子表达中的作用。
Objective to investigate the effects of oxidized low-density lipoprotein receptor 1 (LOX-1) on secretion of adhesive molecules mediated by ox-LDL in human umbilical endothelial cells (HUVECs).
选择素是内皮细胞表达的粘附分子选择素家族的一员，介导白细胞与内皮细胞粘附，可以在动脉粥样硬化病变中发现其表达。
P-selectin is one of the selectin family members, which can result in the adhesion between leucocytes and endothelial cells. It has been detected in atherosclerosis plaques.
近年来，有研究提出内源性甲醛（FA）可能是启动内皮细胞损伤，导致动脉粥样硬化的一个潜在危险因素。
Recently, it was proposed that endogenous formaldehyde(FA) may be involved in endothelial damage, and may be a potential factor of vulnerability of atherosclerosis.
目的观察胰岛素样生长因子结合蛋白1对人血管内皮细胞的影响及其对胰岛素样生长因子的调节作用。
Objective to study the biological activity of the human insulin like growth factor binding protein 1 (IGFBP 1) and insulin like growth factor (IGF 1) on endothelial cell proliferation.
本研究采用流室系统，定量研究了动脉粥样硬化性脑梗塞患者红细胞与内皮细胞粘附特性。
Adherence of erythrocytes to endothelial cells in patients with atherosclerotic cerebral infarction was quantitatively studied by using the flow chamber.
血管内皮细胞乙酰胆碱作用靶标(ETA)作为特异性血管内皮细胞药物作用新靶点具有良好的抗动脉粥样硬化应用前景。
Endothelial target for acetylcholine (ETA) plays an important role in preventing atherosclerosis, and it may be a unique molecular target of the novel drugs against atherosclerosis.
目的：研究血管内皮细胞在致动脉粥样硬化因子作用下，差异表达基因的结构与功能。
Objective: to study the differences of expressed genes in vascular endothelial cells with the action of atherogenic factors and to explore the structure and function of these genes.
NO自1986年在内皮细胞中的重要作用被发现以来，越来越受人们重视，并成为动脉粥样硬化发病机制中的一个新的研究热点。
People has paid more and more attention to NO since its important function in endothelium was found in 1986, and it has become a new hot research point of the pathogenesis of atherosclerosis.
目的探讨人类巨细胞病毒(HCMV)感染、肿瘤坏死因子(TNF)及血清内皮素(et)浓度与动脉粥样硬化的关系。
Objective to investigate the relationship between human cytomegalovirus (HCMV) infection, tumor necrosis factor (TNF), endothelin et in patients with coronary heart disease.
目的克隆、分离血管内皮细胞在致动脉粥样硬化因素作用下差异表达的基因，了解动脉粥样硬化发生的分子机制。
Objective to isolate and clone the differential expressed genes induced by atherogenic factors on vascular endothelium and to understand the molecular mechanism of atherosclerosis.
内皮细胞损伤和单核细胞黏附到血管内皮细胞是动脉粥样硬化发生的重要始动环节。
Monocyte adhesion to endothelial cells is an important initial event at the onset of atherosclerosis.
PDGF有利于三维培养内皮细胞在有展性的胶原基质面形成血管样结构。
Three dimension cell culture found PDGF could induce endothelial cell angiogenesis in vitro.
跟EPO一样，血管内皮生长因子促使红细胞的形成从而帮助组织供血。
Like EPO, vascular endothelial growth factor spurs red-blood-cell formation and thus helps to supply tissues with oxygen.
血管内皮细胞在抗动脉粥样硬化、抗血栓、溶血栓和舒张血管方面具有重要作用。
Endothelial cells of blood vessel play an important role in anti-atherosclerosis, antithrombosis, thrombolysis and vessel dilation.
管壁内皮细胞肿胀，似鞋钉样突向管腔。
Strut of cell of tube wall endodermis, appear hobnail kind dash forward to tubal antrum.
此因子在正常动脉中不表达，但在血管成型术后或者动脉粥样硬化引起的内皮细胞损伤时表达量都有所增加。
This factor was not expressed in normal arteries, but expression of PDGF has increased after angioplasty or atherosclerosis induced endothelial cell injury.
本发明的材料和方法包 含细胞，优选内皮细胞或具有内皮样表型的细胞。
The materials and methods of the present invention comprise cells, preferably endothelial cells or cells having an endothelial-like phenotype.
其中用于提高耐力的主要有促红细胞生成素、血管内皮生长因子等，用于提高肌肉力量的有胰岛素样生长因子-I、肌肉生长抑制素、生长激素、机械生长因子等。
In those gene doping, EPO and VEGF are used to enhance the endurance performance; IGF-I, myostatin, GH and MGF are used to improve muscle strength.
MMP-2在粥样硬化冠脉斑块中有表达，表达的阳性部位为内膜的血管内皮细胞胞浆、巨噬细胞胞浆、平滑肌细胞胞浆和中膜的平滑肌细胞胞浆。
MMP-2 protein expression can be observed in atherosclerosis, mainly in macrophage, foam cells, endothelial cell, smooth muscle cell in endomembrane, smooth muscle cell and fibrocyre in tunica media.
凝集素样氧化低密度脂蛋白受体(LOX - 1)是内皮细胞膜上特异性识别摄取ox - LDL的最主要受体，LOX - 1在介导ox - LDL导致内皮功能紊乱的过程中起关键作用。
LOX-1 was identified from endothelial cells as the main receptor specially recognizing and uptaking the ox-LDL, LOX-1 plays a key role in the endothelial dysfunction triggered by ox-LDL.
结论：OX - LDL能促进内皮细胞表达MMP - 2，这提示血管细胞外基质的降解在氧化脂蛋白诱发的动脉粥样硬化斑块产生和破裂机制中起着一定作用。
CONCLUSION: OX-LDL enhances the expression and activity of MMP-2 in HUVEC, which may provide a explanation for the phenomenon that OX-LDL induces atherosclerotic lesions and plaque rapture.
山此提示，L-抗坏血酸对内皮细胞的保护作用可能是其延缓动脉粥样硬化进程的关键之一。
These data indicate that the protective role of L-ascorbic acid may retard the pathogenesis of atherosclerosis.
山此提示，L-抗坏血酸对内皮细胞的保护作用可能是其延缓动脉粥样硬化进程的关键之一。
These data indicate that the protective role of L-ascorbic acid may retard the pathogenesis of atherosclerosis.
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