结论：心肌缺血预处理与电针内关通过提高内源性氧自由基清除系统酶的活性，抑制缺血再灌注心肌细胞膜脂质过氧化反应，对缺血再灌注心肌具有明显的保护作用。
Coneclusion:EA at Neiguan point and IC can protect Z/R myocardium evidently by raising the activity of inhabiting cell membrane lipid peroxidation of myocardial suffered from ischemic reperfusion.
结论：心肌缺血预处理与电针内关通过提高内源性氧自由基清除系统酶的活性，抑制缺血再灌注心肌细胞膜脂质过氧化反应，对缺血再灌注心肌具有明显的保护作用。
Coneclusion:EA at Neiguan point and IC can protect Z/R myocardium evidently by raising the activity of inhabiting cell membrane lipid peroxidation of myocardial suffered from ischemic reperfusion.
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