结论：大鼠饥饿后早期确有肠黏膜组织结构受损，发生内毒素移位，同时伴有肠黏膜免疫学屏障受损。
CONCLUSION: the structure of intestinal mucosa is damaged in the earlier stage of starvation in rats, accompanied by endotoxin translocation and dysfunction of intestinal mucosal immune barrier.
目的探讨重症急性胰腺炎(SAP)时血浆细胞因子与肠道屏障损害后肠源性细菌和内毒素移位的关系。
Objective to investigate the relationship between the plasma cytokines and the translocation of intestinal bacteria and endotoxin after gut barrier injury in severe acute pancreatitis (SAP) rats.
目的探讨重症急性胰腺炎(SAP)时血浆细胞因子与肠道屏障损害后肠源性细菌和内毒素移位的关系。
Objective to investigate the relationship between the plasma cytokines and the translocation of intestinal bacteria and endotoxin after gut barrier injury in severe acute pancreatitis (SAP) rats.
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